Erstellt von Miriam Pridgen
vor mehr als 5 Jahre
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Limbic System
Limbic System (Anatomy)
Cingulate Cortex
Commonsense View of Emotion
James-Lange Theory
Cannon-Bard Theory
Schacter-Singer (two-factor) theory
Rapid Target for Stress
Target of Stress Over Time
HPA Axis
Alarm Reaction Stage
Resistance Stage
Exhaustion Stage
Voodoo Deaths
Psychosomatic Illnesses
Long Term HPA Effects
Hypothalamus Function (Aggression)
Stimulation of the VMH (Ventromedial Hypothalamus)
Amygdala Stimulation
Rabies
Focal Seizure (Originating from the Amygdala)
Location of Amygdala
Temporal Lobe Epilepsy
Lesion/Removal of the Amygdala
Kluver-Bucy Syndrome
Agonist
Antagonist
Viewing Fearful Faces =
Urbach-Wiethe Disease
CCK (Cholecystokinin)
CCK Agonist =
GABA (Gamma Amino Butyric Acid)
GABA Antagonist
Generalized Anxiety Disorder
Panic Disorder
Phobias
Obsessive-Compulsive Disorder
Post-Traumatic Stress Disorder
Benzodiazepines
Amygdala and Hypothalamus (Benzodiazepines)
Cortex and Thalamus
(Benzodiazepines)
Proband
Proband study
Concordance Rates
Reuptake
Catecholamines
Major Depressive Disorder
Major Depressive Disorder (Genetic Evidence)
Happy mood elicits
Depressed mood elicits
Left Hemisphere Damage
Right Hemisphere Damage
Tricyclics
SSRIs (Selective Serotonin Reuptake Inhibitors)
MAOIs (Monoamine Oxidase Inhibitors)
Electroconvulsive Therapy (ECT)
Depressed Patients
SAD Patients
Schizophrenia Stats
Neuroanatomical Hypothesis of Schizophrenia
Smaller in Schizophrenics
Larger in Schizophrenics
Schizophrenia Lower Activity
Schizophrenia Larger Areas of Brain
Dopamine Hypothesis of Schizophrenia
Dopamine Hypothesis of Schizophrenia (Supporting Evidence)
Glutamate Hypothesis of Schizophrenia
Glutamate Hypothesis of Schizophrenia (Supporting Evidence)