Erstellt von Danielle Morley
vor mehr als 10 Jahre
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At what age does the posterior dominant rhythm begin to appear? What frequency is it seen to be at this time?
At what age should the posterior dominant rhythm reach 8Hz?
What is the normal frequency for the posterior dominant rhythm in adults?
Can a posterior dominant rhythm of less than 8Hz or less be considered normal? If so, in which circumstances?
Can an asymmetrical posterior dominant rhythm be considered normal? If so, explain. Also what montage should be used to determine amplitude asymmetries?
Can alpha be abnormal? If so, when?
Can it be normal for alpha to not be reactive to eye opening? Explain.
When is the best time to review the frequency of the posterior dominant rhythm? Why is this?
At what age do sleep spindles begin to appear? Describe what they look like at this point.
By what age should sleep spindles be symmetrical? By what age should they be fully synchronous?
In a 1yr old patient, sleep spindles are seen only on the right side. Is this normal or abnormal? Why or why not?
In what state(s) are spindles seen?
Name the following pattern. What prognosis does this pattern hold? -Diffuse, non reactive, anterior max alpha
Name the following pattern. What prognosis does this pattern hold? -Prominent, non-reactive, continuous spindle-like activity.
Name indications of drowsiness on the EEG.
What does POSTS stand for? What does this pattern indicate? At what age is it seen?
What is a pattern that is similar to POSTS? Under which conditions is this pattern seen? Is this normal or abnormal?
What are some normal variants that mimic epileptiform activity?
Name the following pattern. What condition(s) may this pattern be associated with? What prognosis does this pattern hold? -generalized sharp or tripasic discharges that are seen periodically @1-2 times per second
Name the following pattern. What condition(s) may be associated with this pattern. What prognosis does this pattern hold? -very high amplitude polyphasic mixed sharp and complexes. Complexes are stereotyped and periodic, occuring every 8 seconds. Myoclonic jerks are associated with the bursts. Underlying background is of normal amplitude, moderately abnormal.
Give definition and clinical description of syncope. Also include common causes or triggers.
What EEG changes (if any) are seen during and after presyncope and syncope?
What is convulsive syncope? What causes it? What will an EEG during convulsive syncope show? How can it be differentiated form true seizure activity (ie GTC)?
Give a clinical description of migraine. What causes migraines?
What EEG changes (if any) are seen with migraines?
What is Moyamoya disease? Where did it get it's name? What population has the highest incidence of the disorder? Why it relevant to EEG?
What is Alzheimer's disease? What are the classic symptoms? What is the typical age of onset? What is the prognosis? What are the pathological findings/ causes?
What are the EEG findings in Alzheimer's disease?
What is Parkinson's disease? What are the classic symptoms? What is the typical age of onset? What is the prognosis? What are the pathological findings/ causes? What does typical treatment consist of?
What are the EEG findings in Parkinson's disease?
What is Lewy Body disease? What are the pathological findings/ causes? What are the symptoms? What age group does it usually affect? What is the prognosis?
What are the EEG findings in Lewy Body disease?
What is Picks disease? What are the pathological findings? What is the typical age of onset? How does someone get this disease? What is the prognosis? What are the classic symptoms?
What are the findings in Pick's disease?
What is meningitis? Where does it come from/what causes it? What are the key symptoms? What are some other symptoms? What kind of outcome/prognosis is expected with meningitis? What factors affect prognosis and how so? What are some common long term issues that arise as a result of meningitis?
What are symptoms seen with meningitis- give classic triad then additional symptoms. Also give symptoms in infants.
What are the EEG changes in bacterial meningitis? Viral?
Is there a time when a cerebral lesion may not produce an abnormality on the EEG? If so, list possible cases where this might be true.