transfusion reactions= type II

autoantigens= type II

delayed hypersensitivy (delayed)

production of granulomas

contact sensitivity (type IV)

tuberculin reaction= type IV

hypersensitivity

three factors leading to development of HS

type I immediate IgE mediated, allergies

type I sensitisation phase

type I effector phase

tx of allergy

type II sensitivity IgG and IgM mediated aka cytotoxic

rhesus incompatibility= type II