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The term used to describe a collection of mechanisms that influence the active and changing circulation blood is
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perfusion
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cardiac output
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stroke volume
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hemodynamics
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Blood flows because of
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a pressure gradient
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Fick's formula
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inotropic factors
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the ejection fraction
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Determining the cardiac output is usually accomplished by
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using Fick's formula
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using Starling's law
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calculating SV X CR = CO
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using Poiseuille's law
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Which of the following has the ability to alter heart rate
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chonotropic factors
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baroreceptors
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carotid sinus reflex
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all of the above
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A change in heart rate or stroke volume does not always change the heart's output, the amount of blood in the arteries, or the blood pressure
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If blood pressure within the aorta or carotid sinus suddenly increases beyond the set point, the control center will increase vagal inhibition and return the blood pressure to normal.
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The amount that the CO can increase above the resting value is called the inotropic factor.
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The ejection fraction is related to the stroke volume
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Peripheral resistance in arteries determines arterial blood pressure
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[blank_start]Viscocity[blank_end]: thickness of blood
[blank_start]Vasoconstriction[blank_end]: reduction in vessel diameter
[blank_start]Perfusion[blank_end]: flow through
[blank_start]Cardiac Output[blank_end]: amount of blood that flows out of a ventricle of the heart per unit of time
[blank_start]Hypoxia[blank_end]: deficiency of blood oxygen
[blank_start]Ischemic[blank_end]: inadequate blood supply
[blank_start]Active hyperemia[blank_end]: local vasolidation
[blank_start]Contractility[blank_end]: the ability of a muscle cell to shorten to product movement
[blank_start]Hypercapnia[blank_end]: excess carbon dioxide
[blank_start]Vasometer pressure flex[blank_end]: inititiated by a change in arterial blood pressure
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Viscocity
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vasoconstriction
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cardiac output
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hypoxia
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vasomotor pressure flex
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hypercapnia
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active hypermia
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ischemic
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perfusion
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contractility
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Vasoconstriction
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contractility
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perfusion
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ischemic
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active hypermia
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hypercapnia
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hypoxia
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vasomotor pressure flex
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viscocity
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cardiac output
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Perfusion
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cardiac output
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vasoconstriction
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viscocity
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vasomotor pressure flex
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hypoxia
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hypercapnia
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active hypermia
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ischemic
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contractility
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Cardiac Output
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vasoconstriction
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viscocity
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vasomotor pressure flex
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hypoxia
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hypercapnia
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active hypermia
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ischemic
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perfusion
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contractility
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Hypoxia
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vasoconstriction
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cardiac output
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viscocity
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vasomotor pressure flex
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hypercapnia
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active hypermia
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ischemic
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perfusion
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contractility
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Ischemic
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contractility
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perfusion
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active hypermia
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hypercapnia
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hypoxia
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vasomotor pressure flex
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viscocity
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cardiac output
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vasoconstriction
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Active hyperemia
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vasoconstriction
-
cardiac output
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viscocity
-
vasomotor pressure flex
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hypoxia
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hypercapnia
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ischemic
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perfusion
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contractility
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Contractility
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perfusion
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ischemic
-
active hypermia
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hypercapnia
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hypoxia
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vasomotor pressure flex
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viscocity
-
cardiac output
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vasoconstriction
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Hypercapnia
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vasoconstriction
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cardiac output
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viscocity
-
vasomotor pressure flex
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hypoxia
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active hypermia
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ischemic
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perfusion
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contractility
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Vasometer pressure flex
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vasoconstriction
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cardiac output
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viscocity
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hypoxia
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hypercapnia
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active hypermia
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ischemic
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perfusion
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contractility
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The ability of blood vessels to expand and adapt to higher pressure and maintain normal flow is called the [blank_start]stress[blank_end] [blank_start]relaxation[blank_end] [blank_start]effect[blank_end].
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Increased respirations and increased [blank_start]circulation[blank_end] tend to coincide.
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[blank_start]Capillary[blank_end] [blank_start]exchange[blank_end] is the exchange of materials between plasma in the capillaries and the surrounding interstitial fluid of the systemic tissues.
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[blank_start]Osmotic[blank_end] [blank_start]pressure[blank_end] tends to promote diffusion of fluid into the plasma.
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The more ADH that is secreted, the more water will be [blank_start]extracted[blank_end] into the blood from the urine and the greater the blood plasma volume will become.
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[blank_start]Renin[blank_end] [blank_start]Angiotensin[blank_end] [blank_start]Aldosterone[blank_end] [blank_start]System[blank_end] of aldosterone secretion changes blood plasma volume.
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Renin
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Angiotensin
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Aldosterone
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System
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[blank_start]ANH[blank_end] is secreted by specialized cells in the atrial wall in response to overstretching.
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[blank_start]Hypertension[blank_end] is high blood pressure.
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The diastolic blood pressure is
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With a blood pressure of 120/80, the number 80 indicates
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the diastolic reading
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they systolic reading
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the Korotkoff sounds
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the ejection phase
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The mean arterial pressure MAP for a BP of 130/90 is
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[blank_start]Sphygomomanometer[blank_end] is the apparatus used to measure blood pressure.
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If blood gushes forth in spurts with considerable force, you have most likely cut an [blank_start]artery[blank_end].
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The pressure points can be used to stop [blank_start]arterial[blank_end] bleeding.
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Starling's law of the heart states that
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blood flows from areas of high pressure to areas of low pressure
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the volume of blood ejected from the ventricle is constant
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the more stretched the heart fibers are at the beginning of a contraction, the stronger is their contraction
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average heart rate is 72 beats per minute
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The vagus nerve is said to act as a [blank_start]brake[blank_end] on the heart.
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brake
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temperature monitor
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positive feedback loop
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ejection mechanism
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Under normal conditions, blood viscosity changes
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frequently
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during hemorrhage only
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under stress
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very little
Frage 28
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The popliteal pulse point is found
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Peripheal resistance is primarily affected by
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the length of myocardial fibers
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blood viscocity and the diameter of arterioles
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the capacity of the blood reservoirs
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the elasticity of the heart
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Septic shock is caused by
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Hypovolemic shock is caused by
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The shift of the blood reservoir to the veins in the legs when standing is called the
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Fick's formula is used to determining
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stroke volume
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cardiac output
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cardiac reserve
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ejection fraction
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The minute volume is equal to the
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the pressure gradient divided by the resistance
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mean arterial pressure divided by the cardiac output
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cardiac output divided by the ejection fraction
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cardiac output divided by the cardiac reserve
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The chief determinant of arterial blood pressure is the [blank_start]volume[blank_end] of blood in the arteries.
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Factors that affect the strength of myocardial contraction and, therefore, stroke volume are [blank_start]inotropic[blank_end] factors.
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Starling's law of the heart states that within limits, the longer, or more stretched, the heart fibers art the beginning of contraction, the [blank_start]stronger[blank_end] is their contraction.
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The [blank_start]ejection[blank_end] [blank_start]fraction[blank_end] is the ratio of the stroke volume to the end-diastolic volume (EDV).
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The pumping work that the heart must do to push blood into the arteries is known as [blank_start]cardiac[blank_end] [blank_start]afterload[blank_end].
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The hormone most known as a heart accelerator is [blank_start]epinephrine[blank_end].
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During exercise blood from reservoirs is redistributed to more active structures such as [blank_start]skeletal[blank_end] muscles and the heart.
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The blood vessel commonly used to perform blood pressure readings is the [blank_start]brachial[blank_end] [blank_start]artery[blank_end].
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The sounds made during the measurement of a blood pressure are called [blank_start]Korotkoff[blank_end] [blank_start]sound[blank_end].
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Blood flows most rapidly in the [blank_start]arteries[blank_end] and most slowly in the [blank_start]capillaries[blank_end].