L26 Clinical pharmacology of hypertension

Descripción

PHCY310 Test sobre L26 Clinical pharmacology of hypertension, creado por Mer Scott el 14/04/2019.
Mer Scott
Test por Mer Scott, actualizado hace más de 1 año
Mer Scott
Creado por Mer Scott hace más de 5 años
5
0

Resumen del Recurso

Pregunta 1

Pregunta
90% of hypertension is ‘primary’ which means of [blank_start]unknown[blank_end] cause. 'Secondary’ is due to kidney disease, Cushing syndrome, thyroid disease, and drug-induced. Drug treatments include drugs to: [blank_start]dilate[blank_end] blood vessels, reduce [blank_start]constriction[blank_end] of vessels, reduce blood [blank_start]volume[blank_end], and reduce cardiac load.
Respuesta
  • unknown
  • dilate
  • constriction
  • volume

Pregunta 2

Pregunta
If we give a Beta1 blocker then we anticipate a decrease in heart rate.
Respuesta
  • True
  • False

Pregunta 3

Pregunta
If we give an alpha blocker then we anticipate vasoconstriction.
Respuesta
  • True
  • False

Pregunta 4

Pregunta
Non-selective β−blockers (e.g. Propanolol) should be avoided in asthmatics as they may constrict the airways.
Respuesta
  • True
  • False

Pregunta 5

Pregunta
The RAAS system maintains fluids and electrolyte balances. Renin is released from the arterioles near the [blank_start]glomerulus[blank_end], in response to reduced [blank_start]blood pressure[blank_end] or reduced renal blood [blank_start]flow[blank_end]. Renin: - Converts [blank_start]angiotensinogen[blank_end] to [blank_start]angiotensin[blank_end] - Angiotensin is converted to [blank_start]angiotensin II[blank_end] by the [blank_start]ACE[blank_end] ATII causes [blank_start]vasoconstriction[blank_end] and aldosterone release from the adrenals. Aldosterone promotes [blank_start]K+[blank_end] excretion in exchange for [blank_start]Na+ and water[blank_end] reabsorption to maintain plasma volume and cardiac output (ie to retain volume and raise BP.)
Respuesta
  • glomerulus
  • blood pressure
  • flow
  • angiotensin
  • angiotensinogen
  • angiotensin II
  • ACE
  • vasoconstriction
  • K+
  • Na+ and water

Pregunta 6

Pregunta
If we give an ACE inhibitor or an angiotensin II receptor blocker (ARB) we anticipate vasodilation.
Respuesta
  • True
  • False

Pregunta 7

Pregunta
Side effects of ACE inhibitors and ARBs: - dizziness/postural [blank_start]hypotension[blank_end] - [blank_start]hyper[blank_end]kalaemia (increase K+) - ACEI can cause [blank_start]cough[blank_end] due to build up of bradykinin (broken down by ACE) [blank_start]Hypotension and hyperkalemia[blank_end] are also side effects of thiazide diuretics.
Respuesta
  • hypotension
  • hyper
  • hypo
  • cough
  • Hypotension and hyperkalemia

Pregunta 8

Pregunta
Which of these is not a side effect of calcium channel blockers?
Respuesta
  • Peripheral oedema
  • Constipation
  • Dizziness/postural hypotension
  • Flushing
  • Headache
  • Nausea

Pregunta 9

Pregunta
Non-dihydropyridine calcium channel blockers can cause bradycardia (Caution if patient also on a β-blocker).
Respuesta
  • True
  • False
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