Pharmacology VI

Question

Quinidine is principally used to treat

Answer 1

a. Hypertension

Answer 2

b. Angina pectoris

Answer 3

c. Congestive hear failure

Answer 4

d. Supraventricular tachyarrhythmias

Answer 5

a. Hypertension

Answer 6

b. Angina pectoris

Answer 7

c. Atrial fibrillation

Answer 8

d. Ventricular fibrillation

Answer 9

e. Congestive hear failure

Answer 10

a. Atrial fibrillation

Answer 11

b. Atrial tachycardia

Answer 12

c. Ventricular tachycardia

Answer 13

d. Catecholamine-induced arrhythmias

Answer 14

a. Digitalis

Answer 15

b. Quinidine

Answer 16

c. Propranolol

Answer 17

d. Procainamide

Answer 18

e. Pentobarbital

Answer 19

a. Digitalis

Answer 20

b. Propranolol

Answer 21

c. Nitroglycerin

Answer 22

d. Isosorbide dinitrate

Answer 23

e. Pentaerythritol tetranitrate

Answer 24

a. Ephedrine

Answer 25

b. Reserpine

Answer 26

c. Methyldopa

Answer 27

d. Thiazide diuretics

Answer 28

a. Atrial fibrillation

Answer 29

b. Congestive heart failure

Answer 30

c. Paroxysmal atrial tachycardia

Answer 31

d. All of the above

Answer 32

a. Digitalis

Answer 33

b. Lidocaine

Answer 34

c. Phenytoin

Answer 35

d. Procainamide

Answer 36

e. Aminophylline

Answer 37

a. Reserpine

Answer 38

b. Guanethidine

Answer 39

c. Phenobarbital

Answer 40

d. Chlorothiazide

Answer 41

e. Alpha-methyldopa

Answer 42

a. Sedatives and reserpine

Answer 43

b. Thiazide diuretics and reserpine

Answer 44

c. Sedatives and thiazide diuretics

Answer 45

d. Guanethidine and ganglionic blocking agents

Answer 46

a. Nadolol

Answer 47

b. Timolol

Answer 48

c. Metoprolol

Answer 49

d. Propranolol

Answer 50

a. Stimulating the beta-adrenergic receptor

Answer 51

b. Suppressing cardiac ATP-ase activity

Answer 52

c. Increasing ectopic pacemaker activity

Answer 53

d. Increasing the refractory period of cardiac muscle

Answer 54

a. Blocking Purkinje fibers

Answer 55

b. Blocking the alpha-adrenergic receptor

Answer 56

c. Suppressing SA node impulse formation

Answer 57

d. Causing a positive inotropic effect

Answer 58

e. Increasing the refractory period of cardiac muscle

Answer 59

a. Blockade of the vagus nerve

Answer 60

b. Stimulation of cardiac ATP-ase activity

Answer 61

c. Blockade of the Beta-adrenergic receptor

Answer 62

d. Stimulation of the beta-adrenergic receptor

Answer 63

e. Increased refractory period of cardiac muscle

Answer 64

a. Increasing AV conduction

Answer 65

b. Decreasing cardiac excitability

Answer 66

c. Increasing cardiac conduction velocity

Answer 67

d. Increasing spontaneous pacemaker activity

Answer 68

a. Abolish cardiac decompensation

Answer 69

b. Inhibit vagal impulses to the heart

Answer 70

c. Decrease the rate of A-V conduction

Answer 71

d. Increase the rate of cardiac repolarization

Answer 72

e. Produce a decrease in the rate of atrial contraction

Answer 73

a. Decreasing the heart rate reflexly

Answer 74

b. Increasing the metabolic work of the myocardium

Answer 75

c. Direct action on smooth muscle in the vessel walls

Answer 76

d. Increasing the effective refractory period in the atrium

Answer 77

e. Blocking beta-adrenergic receptors

Answer 78

a. Has a direct action on vascular smooth muscle

Answer 79

b. Blocks autoregulatory mechanisms in the heart

Answer 80

c. Inhibits oxygen metabolism in cardiac cells

Answer 81

d. Provides relief within seconds of an acute anginal attack

Answer 82

e. Prevents chronotropic responses to endogenous epinephrine emotions and exercise

Answer 83

a. Anti-inflammatory effects

Answer 84

b. Antihistaminic effects

Answer 85

c. Increased blood pressure

Answer 86

d. Increased heart rate

Answer 87

e. A sedative effect

Answer 88

a. Serotonin

Answer 89

b. Angiotensin I

Answer 90

c. Angiotensin III

Answer 91

d. Bradykinin metabolites

Answer 92

a. A sedative effect

Answer 93

b. Increased heart rate

Answer 94

c. Increased blood pressure

Answer 95

d. Antihistaminic effects

Answer 96

e. Anti-inflammatory effects

Answer 97

a. Increase renal excretion of sodium and chloride

Answer 98

b. Increase renal excretion of potassium

Answer 99

c. Increase the toxicity of digitalis

Answer 100

d. Exacerbate existing diabetes

Answer 101

e. Cause hypokalemia

Answer 102

f. Cause hypoglycemia

Answer 103

a. Increasing the glomerular filtration rate

Answer 104

b. Decreasing the renal reabsorption of sodium

Answer 105

c. Decreasing the renal excretion of chloride

Answer 106

d. Increasing the renal reabsorption of potassium

Answer 107

e. Increasing the secretion of antidiuretic hormone

Answer 108

b. Chlorothiazide

Answer 109

c. Theophylline

Answer 110

d. digitalis glycosides

Answer 111

e. Procainamide

Answer 112

a. Activation of adenylcyclase

Answer 113

b. Inhibition of phosphodiesterase

Answer 114

c. An agonist effect of beta-receptors

Answer 115

d. Inhibition of Na+, K+ ATPASE leading to increased calcium influx

Answer 116

e. Decreasing the amount of calcium available for excitationcontraction coupling

Answer 117

a. Is primarily a diuretic

Answer 118

b. Reduces the ventricular rate

Answer 119

c. Decreases abnormal cardiac rhythms

Answer 120

d. Produces peripheral vasoconstriction

Answer 121

e. Has a positive cardiac inotropic action

Answer 122

a. Force of contraction

Answer 123

b. Ventricular excitability

Answer 124

c. Refractory period of the atrial muscle

Answer 125

d. Refractory period of the ventricular muscle

Answer 126

e. Rate of conduction of impulse to the muscle

Answer 127

a) (a), (b) and (c)

Answer 128

b) (a) and (c) only

Answer 129

c) (c) and (d)

Answer 130

d) (e) only

Answer 131

e) All of the above

Answer 132

b. Bromide

Answer 133

c. Calcium

Answer 134

d. Chloride

Answer 135

e. Potassium

Answer 136

b. Lithium

Answer 137

c. Calcium

Answer 138

d. Chloride

Answer 139

e. Magnesium

Answer 140

b. Urine flow

Answer 141

c. Heart size

Answer 142

d. Heart rate

Answer 143

e. Residual diastolic volume

Answer 144

a. Block beta-adrenergic receptors

Answer 145

b. Inhibit formation of angiotensin II

Answer 146

c. Inhibit nerve-induced release of norepinephrine

Answer 147

d. Stimulate central inhibitory alpha-adrenergic receptors

Answer 148

e. Inhibit the postsynaptic of norepinephrine on vascular smooth muscle

Answer 149

a. Methyldopa

Answer 150

b. Metoprolol

Answer 151

c. Hydralazine

Answer 152

d. Propranolol

Answer 153

e. Guanethidine

Answer 154

a. Prazosin

Answer 155

b. Clonidine

Answer 156

c. Propranolol

Answer 157

d. Guanethidine

Answer 158

e. Chlorothiazide

Answer 159

a) (a) and (b)

Answer 160

b) (a) and (d)

Answer 161

c) (b), (c) and (d)

Answer 162

d) (c), (d) and (e)

Answer 163

e) (c) and (e) only

Answer 164

a. Sedation

Answer 165

b. A diuretic effect

Answer 166

c. An antirenin effect

Answer 167

d. A vagal blocking effect

Answer 168

e. An increase in cardiac output

Answer 169

a. Glycogenolysis

Answer 170

b. Increased cardiac output

Answer 171

c. Decreased diastolic pressure

Answer 172

d. Decreased peripheral resistance

Answer 173

e. Relaxation of bronchial smooth muscle

Answer 174

a. Osmotic

Answer 175

b. Thiazide

Answer 176

c. Mercurial

Answer 177

d. High-ceiling

Answer 178

a. Osmotic

Answer 179

b. Thiazide

Answer 180

c. Mercurial

Answer 181

d. High-ceiling

Answer 182

a. Extrasystoles

Answer 183

b. Nausea and vomiting

Answer 184

c. Yellow-green vision

Answer 185

d. A-V conduction block

Answer 186

d. Chlorothiazide

Answer 187

e. Acetylsalicylic acid

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Pharmacology VI

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