707442
Pregunta 1
Pregunta
Which of these is not a type of cel death?
Respuesta
-
Apoptosis
-
Necrosis
-
Autolysis
-
Oncosis
-
utiss
Pregunta 2
Pregunta
Which of these is not part of apoptosis?
Respuesta
-
Membrane integrity preserved
-
Active processes, requires ATP and protein syntehsis'caspase cascade activated
-
cells shrink and components packaged into membrane-bound vesicles (apoptic bodies)
-
Single cels
-
no inflammation
-
Pasive process
Pregunta 3
Pregunta
Which of these is not part of oncosis?
Respuesta
-
Membranes breached
-
Passive process,driven by energy deprovation
-
Cells swell with haphazard destruction of organelles
-
contiguous cells
-
inflammatory response
-
active process
Pregunta 4
Pregunta
Which are the causes of cell damage?
Respuesta
-
Oxygen deficiency(hypoxia)
-
Physical agents
-
chemicals and drugs
-
Micro-organisms
-
Immune reactions
-
creams
Pregunta 5
Pregunta
Which of these causes ischaemia heart conditions?
Respuesta
-
Angina pectoris
-
MI
-
Sudden cardiac death
-
Chronic ischaemic heart disease
-
endocardidit s
Pregunta 6
Pregunta
What are the complications of MI?
Respuesta
-
Reduced Myocardial contractility --> acute heart failure
-
Arrhythmias and heart block
-
papillary muscle dysfunction
-
Myocardial rupture
-
Change in blood group
-
pericarditis
-
mural thrombus
-
ventricular aneurysm
-
distorted papillary muscles
Pregunta 7
Pregunta
Dressler syndrome is an autoimmune complication after an MI, caused by immune system attacking the pericardium.
Respuesta
- True
- False
Pregunta 8
Pregunta
Which of these are the symptoms and signs of Dressler syndrome?
Respuesta
-
chest pain
-
fever
-
pericarditis with effusion
-
pain in left arm
Pregunta 9
Pregunta
What causes vasoconstriction?
Respuesta
-
Increase in oxygen
-
decrease in CO2 and other metabolites
-
Increase in endothelin
-
Increase in SNS stimulation
-
Increase in vasopressin,
-
Increase in angiotensin II
-
Cold
-
Decrease in O2
-
Increase in Co2
Pregunta 10
Pregunta
What causes vasodilation?
Respuesta
-
Decrease in o2
-
Increase in Co2 and other metabolites
-
Increase NO
-
Decrease in SNS stimulation
-
Decrease in histamine stimulation
-
Heat
-
Increase in Co2
Pregunta 11
Pregunta
Coronary cirulation receives 4-5% resting Cardiac output
Respuesta
- True
- False
Pregunta 12
Pregunta
Is cerebral resistance vessels are spared from baroreflex vasoconstriction?
Respuesta
- True
- False
Pregunta 13
Pregunta
Is this correct?
MAP=CO X SVR
Respuesta
- True
- False
Pregunta 14
Pregunta
Which factors affect venous return?
Respuesta
-
CO
-
Muscle pump
-
venous valves
-
thoracic pump
-
blood group
Pregunta 15
Pregunta
Which of these are the types of shock?
Respuesta
-
Hypovolaemic
-
Cardiogenic
-
Obstructive
-
Septic
-
Anaphylactic
-
Neurogenic
-
hypotension
Pregunta 16
Pregunta
Acute coronary syndrome is a result of rupture of an atheromatous plaque?
Respuesta
- True
- False
Pregunta 17
Pregunta
Non vunerable plaques have thin fibrous capsule, large atheromatous core
Respuesta
- True
- False
Pregunta 18
Pregunta
Blood pressure is determined by CO and Peripheral resistance
Respuesta
- True
- False
Pregunta 19
Pregunta
Normal pulse pressure is 40mmHg
Respuesta
- True
- False
Pregunta 20
Pregunta
Mean arterial pressure is 93mmHG
Respuesta
- True
- False
Pregunta 21
Pregunta
Where are baroreceptors located?
Respuesta
-
Carotid sinus
-
aortic arch
-
renal arteries
Pregunta 22
Pregunta
The two types of fibres responding to stretch artery?
Respuesta
-
A fibres
-
C fibres
-
D fibres
Pregunta 23
Pregunta
Which of these are secondary hypertension?
Respuesta
-
Renal hypertension
-
Endocrine tumours -Conn's tumours: primary hyperaldosterone -Phaeochromocytoma:adrenal medulla
-
Coarctation of the aorta
-
pre-eclampsia of pregnancy
-
liquorice addiction
-
flu
Pregunta 24
Pregunta
Primary(essential) hypertension?
Respuesta
-
Genetic -Glutathione-S-transferase -eNOS -B2 adrenoreceptor polymorphism
-
Renal -Hypertension travels with kidneys -reduced nephron number in hypertensives
-
Stomach
Pregunta 25
Pregunta
120/80mmHg is normal
Respuesta
- True
- False
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