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Descripción
Fichas por sammie_church, actualizado hace más de 1 año
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Creado por sammie_church
hace más de 8 años
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| Pregunta | Respuesta |
| Biological explanations of Anorexia Nervosa | By Samantha Church |
| Serotonin A01 | Serotonin is a brain neurotransmitter that is involved in many behavioural functions, including depression and OCD. Early studies found a disruption in the levels of serotonin in people with eating disorders such as AN. This disruption has sometimes been found to be higher levels, and sometimes lower, but all research has shown consistently disturbed levels |
| Serotonin-A01 PET scans | Brain scanning techniques are now being used to look at serotonin in the brain. PET are now able to show the number of different serotonin receptors in different parts of the brain, by injecting a drug which binds to the serotonin receptors and shows up as a brightly lit area on the screen. PET have shown that individuals with AN have fewer serotonin receptors, which is said to even be true of those recovering from AN. |
| Serotonin-A02 SUPPORT (personality traits) | The first supporting factor of serotonin, is the idea that levels of it are linked to personality traits associated with AN such as anxiety, depression and perfectionism. |
| Serotonin-A02 SUPPORT (hypothalamus) | Serotonin is also part of the neurotransmitter system associated with the hypothalamus, which controls eating behaviour. Therefore, disturbed levels of serotonin must have an effect on eating behaviours, such as drastic eating linked with eating disorders like AN. |
| Serotonin-A02 SUPPORT (Bailer et al) | Bailer et al looked at three different groups of women; a control group, those recovering from AN, and those recovering from binge-purge AN. He found the highest levels of serotonin in those recovering from binge-purge AN, and the highest levels of serotonin were found in the individuals with the most anxiety. This therefore shows that persistent disruption of serotonin levels could lead to anxiety which then could lead to AN. |
| Serotonin-A02 CRITICISM | The first criticism is that most of the research into this is carried out with patients with ongoing AN, and so therefore could show a biased view of the relationship between serotonin and AN, and therefore other factors could be involved, so no causal conclusions can be made. |
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