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( Curran, Tauscher, Ripke ) et al (2004) found that dopamine ( agonists, antagonists ) (such as amphetamines, which ( increase, decrease ) the levels of dopamine in the brain) can produce schizophrenic symptoms such as hallucinations. This supports the idea that ( hyperdopaminergia, hypodopaminergia ) plays a role in the development of schizophrenia.
( Tauscher, Curran, Ripke ) et al (2014) found that dopamine ( antagonists, agonists ) (such as antipsychotics., which ( decrease, increase ) the levels of dopamine in the brain) can reduce schizophrenic symptoms, supporting the role of ( hyperdopaminergia, hypodopaminergia ).
However, ( Ripke, Tauscher, Curran ) et al found that some of the genes linked to schizophrenia don't code for dopamine, but rather for ( glutamate, histamine, amino acids ), suggesting dopamine ( is not, is ) the sole cause of schizophrenia.