Mobitz type 1 is non-pathogenic reg -irreg, Type 2 is pathogenic, irreg-irreg
Whole complex is missed, insignificant unless HR <20
AVN disease, inflamm or degenerative, K imbalance, or digoxin
causes an exercise intolerance because HR fails to increase
QRS without P
Tx: correct elec imbalance or underlying cause. If there is an inflam stimulus, consider corticosteroids. Phenytoin in chronic cases (Na channel blocker
P with no QRS
systolic murmur common from V contraction while AV valve open
Large atria with carcadian movement of impulses, or high vagal tone cuase refractory period to be different in different cells
non pathological, exercise resolves, but may be recursor to AF
Ventricular atopic beats
Tx if HR >100, there are runs, or R on T phenomenon. Procainamide infusion, Lidocaine infusion with MgSO4
Cardioversion therapy for Atrial fibrillation
Less successful conversion if >6 duration, underlying cardiac disease exists. Poorer prognosis if there is a pathological murmur with volume overload, if HR >55, or previous treatment was unsuccessful
Oral boluses of Quinidine Sulphate:
22mg/kg via stomach tube q2hr, max 5 doses. Vagolytic and prolongs AP so increases the refractory period.
Risk severe ventricular tachycardia if Vs start responding to every AF
Other Signs of toxicity include depression, D+, colic, muzzle swelling, weakness, laminitis, hypotension and collapse leading to death
Managing SEs:
Stop dosing, give mineral oil to decrease further absorption.
NaHCO3 to increase protein binding of QS,
Lidocaine IV for VT tx if rate >100bpm,
MgSO4 if Tsordes de pointes develops (Ca channel blocker so staibilises membranes)
Electric Shock therapy
Can be attempted under GA if unresponsive to medical therapy.
Use echo examination to determine suitability
Non-Pathological Dysrrhythmias
2'AV block, Sinus Block, Sinus arrhythmia, APC, VPC (1 or 2 in 24hr)
Pathological Dysrrhythmia
3' AV block, Sinus Bradycardia, AF, APC, VPC, VT