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3166822
Chronic adaptations
Description
Victorian Certificate of Education (VCE) High School Mind Map on Chronic adaptations, created by Taylor Lilley on 08/08/2015.
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high school
victorian certificate of education (vce)
Mind Map by
Taylor Lilley
, updated more than 1 year ago
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Created by
Taylor Lilley
over 9 years ago
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Resource summary
Chronic adaptations
Aerobic
Heart
Hypertrophy of myocardium (heart)
^ stroke volume
Resting and sub-max HR decrease
Bradycardia; when HR < 60
Q (Cardiac output) remains fairly stable at max and sub-max
Q can increase at maximal exercise though
Blood vessels
^ in capillarisation of the myocardium and skeletal muscles
More capillaries
Slow twitch muscles have greatest increase
+ve relationship between # of (mitochondria and capillaries)/fibre
+ve relationship between # of capillaries/fibre and muscle fibre diameter
Muscles more efficient at extracting O2
Less blood sent to active muscles at except max intensity
More blood sent to skin
Better thermoregulation
More blood is sent to muscles with higher % of type 1 fibres
aVO2-Diff ^
Blood
^ total blood volume
^ plasma (within days)
Factor in thermoregulation ^
Factor in SV ^
^ in RBC count (haematocrit)
Increase in haemoglobin (oxygen carrier)
^ LIP
Increased lack of lactate removal
Decreased rate of lactate production
Basically delays fatigue
Respiratory adaptions
^ lung volumes (except tidal volumes
^ area of alveolar-capillary membrane
^ pulmonary diffusion
^ efficiency
Ventilation decreases above rest
Muscular adaptions
Increase in O2 uptake
Little to no change in strength or power
Aerobic system contributes more ATP
Muscle structure
Type 1 (slow)
^ oxidative capacity
Fibres increase in size
Type 2 (fast)
Take on characteristics of type 1
Do not become type 1
a-VO2-diff gets more efficient
Major contributor to VO2max ^
^ Myoglobin
Compound that transports O2 to mitochondria
Haemoglobin but inside the muscle
Ability to transport increases
Mitochondria
Site of aerobic respiration
Resynthesises ATP
^ Oxidative enzymes
Related to ^ %VO2max before LIP
^ size and number
^ aerobic ATP resynthesis
Ability to oxidise fats ^
Specifically FFA
Glycogen sparing ^
Ability to aerobically utilise glycogen ^
Glycolysis
Anaerobic
Heart
Thickening of LV wall
^ contraction power
NO change in SV
Muscular Adaptations
^ Substrate storage
ATP
Glycogen
PC
^glycolytic capacity and enzyme activity
Annotations:
Ability to respire without oxygen during anaerobic metabolism
Changes occur in all fibres but primarily type 2
Muscular reactions to resistance training
Neural
Improved neuromuscular pathways
More efficient and forceful contractions
^ motor unit recruitment
^ motor unit firing frequency
^ recruitment of fast twitch fibres
^ motor unit co-ordination
^ co-contraction (agonist/antagonist)
Increased activation of stabilising muscles
Tend to occur within first few weeks of training
Hypertrophy
^ # and size of myofibrils
Muscles built with chains of these
^ contractile proteins
Actin and myosin
No increase in # of muscle fibres, only size
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