Chronic Obstructive Pulmonary Disease UPDATED

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Mind Map on Chronic Obstructive Pulmonary Disease UPDATED, created by Rachel Hollywood on 07/03/2017.
Rachel Hollywood
Mind Map by Rachel Hollywood, updated more than 1 year ago More Less
Brittlyn Burkholder
Created by Brittlyn Burkholder over 7 years ago
Brittlyn Burkholder
Copied by Brittlyn Burkholder over 7 years ago
Rachel Hollywood
Copied by Rachel Hollywood over 7 years ago
Brittlyn Burkholder
Copied by Brittlyn Burkholder over 7 years ago
Brittlyn Burkholder
Copied by Brittlyn Burkholder over 7 years ago
Rachel Hollywood
Copied by Rachel Hollywood over 7 years ago
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Resource summary

Chronic Obstructive Pulmonary Disease UPDATED
  1. Pathophysiology
    1. BRONCHITIS (Blue bloater)
      1. 1. Chronic Inflammation and swelling of bronchial mucosa results in scaring and increased fibrosis of the mucous membranes and hyperplasia of mucous glands and goblet cells.
        1. 2. Leads to thickenening of the bronchial walls
          1. 3. Obstruction of airflow
            1. 4. Increased levels of eosinophils at site of inflammation
              1. 5. Hypertropy of goblet cells leads to increased production of mucous
                1. 6. Mucus production combined with purulent exudate forms a bronchial plug
                  1. Often bacterial conolization contains bacteria of the H. influenza and S. pneumoniae groups
                    1. 7. Narrowed airway and mucous plugs lead to improper oxygenation, thus causing high airway resistance
                      1. 8. Increased oxygen demand (Perfusion and ventialation mismatch)
                        1. 9. Can lead to oxygen desaturation (cyanosis) due to the inabiltity to the alveoli exchanging CO2 for O2 slowly. This can lead to an abundance of alternative issues.
                          1. Can also lead to right sided heart failure and edema
          2. (Schumann, 2013, p. 482)
          3. EMPHYSEMA (Pink Puffer)
            1. 1. Reductions to the capillary bed occur due to smoking, enviromental pollutants and certain occupations.
              1. 2. Destroyed alveolar walls cause inflammation
                1. 3. Inflammation releases protolytic enzymes such as macrophages and neutrophils
                  1. 4. This loss of capillary bed causes reduced pulmonary abilities and causes the loss of elasticity
                    1. 5. Loss of elasticity leads to less exchange of gas and a increasing pressure around the airway lumen
                      1. 6. Leads to airway resistance and decreased airflow
                        1. 7. Air becomes trapped in distal alveoli and they become distended which can collapse with the pressure from airway obstruction
              2. (Schumann, 2013, p. 487)
              3. Clinical Manifestations of COPD: Intermittent or productive cough, sputum production, dyspnea
                1. DIAGNOSTIC: COPD is diagnosed based on risk factors and symptoms. COPD is confirmed by a spirometry test. This allows COPD to be classified from mild to very severe. Specifcally, the FEV ratio of less than 70% indicates COPD.
                  1. ACUTE EXACERBATION OF CHRONIC OBSTRUCTIVE PULMONARY DISEASE
                    1. COLLABORATIVE PRACTICE
                      1. There are 7 goals associated with managing AECOPD - Prevention progression - Reduce the frequency of exacerbation phases - Relieve breathlessness and respiratory symptoms - Improve exersise and daily activities - Treat exacerbation - Improve quality of life and health status - Reduce risk of mortality (Lewis, Heitkemper, Dirksen, Bucher, & Camera, 2014)
                        1. someone with COPD mid 80s- low 90%. Oxygen can be dangerous and lead to further complications -Increased risk of hypoxemia due to lack of ability to effectively exchange CO2 for O2.
                          1. Signs of Hypoxemia : Skin colour changes, Shortness of breath, confusion, wheezing, rapid heart rate, and rapid breathing.
                          2. Smoking Cessation is a large focus for those with COPD and also those that are excperincing exacerbation. 80-90% of those with COPD were at one time a smoker. Smoking leads to an accelerated rate of pulmonary function decline. Thus causing a increased risk of exacerbation and risk of severe complications. (Lewis, Heitkemper, Dirksen, Bucher, & Camera, 2014)
                            1. During exacerbation an individuals body can become decompensated due to the extreme conditions; improving this by ensuring adequate nutrition, exercise and deep breathing and coughing.
                              1. In severe case of AECOPD surgical therapy many be indicated; -Lung volume reducation surgery; reduces the size of the hyper nflated emphysematous lungs by 20-25% - Lung transpalant if advanced.
                                1. Drug Therapy A crucial part of managing AECOPD. The use of oral and parenteral corticosteroid use is a rapid methode that reduces the risk of relapse. (Carr, 2008) -A corticosteroid in AECOPD improves airflow and will recuce the likeliness of alternative treatment failures (Woods, Wheeler, Finch, & Pinner, 2014) - During a exacerbation period it often leads to a increased amount of drugs being used, and new ones being introduced this is to help quickly manage the symptoms and seek a new regimen.
                                  1. Ensure that client has recieved all vaccinations and their annual influenza and pneumoccacal vaccine
                                    1. Avoid exposures to large crowds in peak of influenza season
                                    2. Oxygen is a drug that should be used with caution
                                2. THERAPEUTIC NURSING CARE
                                  1. HEALTH PROMOTION
                                    1. Avoid exposure to work and environmental pollutants and irratants
                                      1. Hand hygiene
                                        1. Avoid or quit smoking
                                          1. Avoid large crowds during peak influenza seasons
                                            1. Vaccinations
                                              1. Seek medical attention when change in baseline
                                                1. Early detection of airway disease and respiratory tract infections
                                              2. EDUCATION
                                                1. Nonpharmacological therapy: breathing exercises, pursed lip breathing, relaxation techniques, exercise.
                                                  1. What is COPD? Medications and correct use of inhalation devices. Home oxygen.
                                                    1. Psychosocial/Emotional Issues: dependency, intimacy, depression, anxiety, panic, support, rehabilitation groups.
                                                      1. End of Life: identifying concerns, support decision making and planning
                                                        1. COPD Management
                                                          1. It is important to have a written action plan, which will include monitoring signs and symptoms. Immediately reporting changes and recongnizing signs and symptoms of infection are important in mangaging COPD. The following is a resource used to plan the car e of COPD. http://www.respiratoryguidelines.ca/sites/all/files/CTS_COPD_updated_Action_Plan_editable_PDF.pdf
                                                          2. NUTRITION High protein and high calorie diets are encouraged, as the high calorie and protein prevent weight loss and muscle wasting. Weight loss can occur due to the lowered energy, increased work of breathing and shortness of breath.
                                                          3. EXERCISE
                                                            1. Energy Conserving Strategies
                                                              1. Strategies include using alternative techniques to complete ADLs. It is reccommended that care that uses the upper extremities, like shaving, hair care, etc. is down sitting down in a tripod posture (elbows being supported), while a mirror is placed on the table in front. It is also important for a patient to have their oxygen on during times of activity to conserve energy. Patients are encouraged to exhale when doing activity.action, and inhale on rest.
                                                              2. Sexual Activity
                                                                1. It is recommended that to help conserve energy, patients with COPD have sexual activity during a time that their breathing is at best. During sexual activity it is important for the patient to practice slow pursed-lip breathing, and to refrain from sexual activity after eating or other strenuous activities.
                                                                2. Sleep
                                                                  1. Psychosocial Considertions
                                                                    1. Patients often have a range of emotions when dealing with a COPD diagnosis or a complication (AE). It is difficult for patients to come to terms with the reality that they are unable to do activities or care for themselves like they did. Nurses are to provide emotional support, and provide therapeutic interventions like relaxation techniques.
                                                                3. Infectious Triggers
                                                                  1. Most common cause of AECOPD
                                                                    1. H. influenza & S.pneumoniae
                                                                    2. Non-infectious Triggers
                                                                      1. Exposure to allergens, irrantants, cold air and air polutions
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