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6914457
Nadia Burns Her Hands
Descrição
Mapa Mental sobre Nadia Burns Her Hands, criado por Menna Emam em 07-11-2016.
Mapa Mental por
Menna Emam
, atualizado more than 1 year ago
Mais
Menos
Criado por
Menna Emam
quase 8 anos atrás
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Resumo de Recurso
Nadia Burns Her Hands
Burns
Types
Thermal
Chemical
Radiology
Electrical
Inhalational
Friction
Complications
Local
Systemic
Degrees
1st Degree
ONLY Epidermis
Pain / Sun burn / Healing occurs
2nd Degree
Epidermis & part of dermis
Pain / Blister / Healing occurs
3rd Degree
Full thickness / Epidermis & Dermis
NO pain / Tissue damage / Healing MIGHT occur ( depends on the severity )
4th Degree
Full thickness , sub-dermal tissue -into- muscles & bones
Painless / Amputation / Functional impairment / Gangrene / Death
Cell Injury
Caused by
Electricity
Radiation
Chemicals
Heat
Signs & symptoms
Redness
Swelling
White or splotchy skin
Waxy or leathery skin
Carbon monoxide poisoning
Blister
Types
Bug bite blister
Friction blister
Cold & heat blister
Hand-foot and mouth blister
Contact Dermatitis
Fever Blister
Virus Blister
Necrosis
Types
Coagulative Necrosis
Liquefactive Necrosis
Gangrenous Necrosis
Caseous Necrosis
Fat Necrosis
Fibrinoid Necrosis
Nuclear changes
Cytoplasmic changes
increased Eosinophilia
Glassy Appearance
Swollen Mitochondria
Bacterial resistance
Resists by
Preventing the antibiotic from getting to its target
Changing the target
Destroying the antibiotic
by Enzymes
Gained by
Getting a copy of gene-encoding an altered protein or an enzyme
How
By scavenging DNA remnants from degraded bacteria
Microbes can join together & exchange DNA to each other
One plasmid can encode resistance to many different antibiotics
Through transposon
Innate Body defense
1st line of defense
Mechanical and chemical barriers
Skin & mucous Membranes
2nd line of defense
Mediated by cells & molecules
Antimicrobial proteins
Interferons
C-proteins
Natural killing cells
Phagocytes
Inflammation
types
Acute
Chronic
Triggered by
Immune reactions
Tissue Necrosis
Trauma
forge in bodies
Infections
Outcome
Healing
Chronic inflammation
changes
Cellular
Vascular
Steps
Removal of agents
Phagocystosis / Bacterial killing
Control the response
Requirement of leukocytes
Adhesion
Transmigration
Chemotaxis
Margination & rolling
Recognition of the injurious agent
PRR
Resolution (Repair)
Fibrosis
Deposition of collagen
Healing of skin wound
By first intention
By second intention
Factors affecting repair
Nutriton
Glucocorticoids
Mechanical variables
Scar formation
Steps
1 Angiogenesis
2. Formation one granular tissue
3. Scars remodel
In case of
Severe injuries
Regeneration
In case of
Superficial injuries
Types of tissue
Stable tissue
Labile tissue
Permanent tissue
Nonspecific defense mechanisms against harmful agents
Burn Infections
Skin & wound cultures
Bacterial , Fungal , Viral
Normal skin flora
Anti-Microbial agents
Bactericidal / Bacteriostatic
Site of action
Classifications
Cell Membrane Function
Metabolic Pathways
Nucleic Acid Synthesis
Protein Synthesis
Cell Wall Synthesis
Psychological Aspects of Burn Patients
Anxiety
Depression
Embarrassment
Helplessness
Anexos de mídia
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