Zusammenfassung der Ressource
Gastritis
- Erosive
- Acute
- Diagnosis
- Endoscopy
- Symptoms
- Bleeding
- hematemesis, melena, or
blood in the nasogastric
aspirate
- Dyspepsia, N/V
- Chronic
- Symptoms
- Few or None
- Dyspepsia, N/V
- Causes
- NSAIDS
- Stress
- ETOH
- Mechanical
Ventilation
- Treat Propholactically
PPI & H2 Blockers
Early Enteral Feeding
- Head Trauma
- Burn Patient
- viral (CMV
- Vascular injury or
Trauma
- Pathophysiology
- Acidic Gastric
erosion of mucosa
- Destruction of
Mucosal Defense
- Treatment
- Endoscopic
Hemostasis
- PPI & H2
Blocker
- IV Fluids & Blood
Transfusion
- Sugery (Fall Back)
- Non-Erosive
- Pathophysicology
- Superficial
- Lymphocytes & Plasma cells w/neutrophils
infiltrate inflammatory cells. Superficial, involve
antrum, body or both. Increases with age
- Deep
- Mononuclear cells & neutrophils infiltrate the
entire mucosa to the level of the muscularis, but
exudate or crypt abscesses seldom result, as
might be expected by such infiltration
- atrophy
- Type A
- Some patients with gastric atrophy have
autoantibodies to parietal cells, usually in
association with corpus (type A) gastritis
and pernicious anemia
- Type B
- Atrophy of gastric glands in gastritis, most
often long-standing antral (sometimes
referred to as type B) gastritis
- Metoplasia
- Mucosal Gland
- (pseudopyloric metaplasia) occurs in the
setting of severe atrophy of the gastric
glands, which are progressively replaced by
mucous glands (antral mucosa), especially
along the lesser curve.
- Intestinal
- begins in the antrum in response to chronic mucosal injury & may
extend to the body. Gastric mucosa cells change to Gastric mucosa
cells change to resemble intestinal mucosa—with goblet cells,
endocrine (enterochromaffin or enterochromaffin-like) cells, and
rudimentary villi—and may even assume functional (absorptive)
characteristics.
- Cause
- Helicobacter pylori infection
- Diagnosis
- Endoscopy for symptomatic
gastritis patients
- Sympoms
- Mild dyspepsia vague symptoms
- Treatment
- H pylori + patients
Eradication of H pylori
- Negative H pylori - patients PPI & H2
blockers or antacids
- Pathophysiology
- Inflamation of
the gastric
mucosa
- Causes
- NSAIDS
- ETOH
- Stress
- AutoImmune
- H. Pylori
- Treatments
- PPI
- H2Blocker
- Antibiotics for
H. pylori
- Prostaglandins
- Sucralfate
- Stoppin the causative actor
- Symptoms
- pain upper
abdom
- Heartburn
- Early Sataity
- Indigetion
- GI bleeding
- Diagnosis
- Symptom Depend
- Endoscopy