Zusammenfassung der Ressource
ventricular septal defects
- type of left to right shunt
- = 30% of all congenital heart disease cases
- defect anywhere in ventricular septum
- perimembranous
- i.e. adj to tricuspid valve
- muscular
- i.e. completely surrounded by mm
- typed according to size
- small VSDs
- smaller than aortic valve diameter
- i.e. up to 3mm
- clinical features
- symptoms
- asymptomatic
- signs
- loud pansystolic
murmur @ lower left
sternal edge
- loud murmur implies smaller defect
- quiet pulmonary second sound P2
- Ix
- CXR
- normal
- ECG
- normal
- inverted T wave
- means no pulmonary HTN
- Echo
- shows anatomy of defect
- Doppler echo- shows haemodynamic effects of VSD
- no pulmonary HTN
- Mx
- close
spontaneously
- murmur will
disappear
- normal ECG on follow up
- normal echo
- maintain
good dental
hygiene
while VSD
present
- prevent bacterial endocarditis
- large VSDs
- defects same
size or bigger
than aortic valve
- clinical features
- symptoms
- after 1 week old
- heart failure
- breathlessness
- failure to thrive
- recurrent
chest
infections
- physical signs
- of heart failure
- tachypnoea
- tachycardia
- hepatomegaly
- active precordium
- soft pansystolic murmur or no murmur
- implies large defect present
- apical mid-diastolic murmur
- from increased flow across
mitral valve after blood has
circulated through the lungs
- loud pulmonary 2nd sound (P2)
- from raised pulmonary arterial Pa
- Ix
- CXR
- cardiomegaly
- enlarged
pulmonary
aa
- increased
pulmonary
vascular
markings
- pulmonary
oedema
- ECG
- biventricular hypertrophy by 2 months of age
- upright T wave
- indicates pulmonary HTN
- Echo
- show anatomy of defect,
haemodynamic effects & pul
HTN (due to high lfow)
- Mx
- heart failure
- diuretics
- captopril
- additional
calorie
input
- surgery
@ 3-6
months
- aims
- manage heart failure & failure to thrive
- prevent lung damage from pulmonary HTN & high blood flow
- kids w/ large VSD & L to R shunt always have HTN
- -> irreversible damage to pulmonary vasc capillary bed