Hemorrhagic Problems in Pregnancy

Description

Nursing (OB NRS 322) Flashcards on Hemorrhagic Problems in Pregnancy, created by Allie Winningham on 01/11/2016.
Allie Winningham
Flashcards by Allie Winningham, updated more than 1 year ago
Allie Winningham
Created by Allie Winningham about 8 years ago
8
2

Resource summary

Question Answer
MAP: mean arterial pressure Normal is 70-110 mmHg. Is an indicator of perfusion of Oxygen and nutrients to vital organs. NEED > 60 mmHg to adequately perfuse vital organs!
MAP is affected by…. Volume of blood ejected from heart/minute, heart rate, blood pressure, and vascular resistance.
MAP = [(2xDiastolic) + systolic] / 3
Antepartum causes of bleeding (before birth) 1st/2nd Trimester: abortion, ectopic pregnancy, gestational trophoblastic disease 3rd: Placenta previa and abruptio placentae
Intrapartum causes of bleeding (during labor) Uterine inversion/rupture, abnormal cord insertions, placental abnormalities.
Postpartum causes of bleeding hemorrhage, retained placental tissue, lacerations, hematomas
Causes of Spontaneous Abortion Chromosomal defects is most common! Teratogens, faulty implantation/fertilization, maternal infection, endocrine disorders (DM and hypothyroidism), anatomic defects (bicornate uterus or incompetent/weak cervix)
Symptoms of Spontaneous Abortion Spotting, light bleeding, uterine cramping, back ache, pelvic pressure (most common before week 12)
DX tests for Spontaneous Abortion Cervical exam, ultrasound, Hematocrit and Hgb, low B-hCG for gestational age, serum progesterone
Treatment of Spontaneous Abortion Abstinence for at least 2 weeks! Sedation, blood transfusion, IV fluids, D&C, Rhogam!
Dilation and Curettage Dilation and curettage (D&C) is a procedure to remove tissue from inside your uterus. Doctors perform dilation and curettage to diagnose and treat certain uterine conditions — such as heavy bleeding — or to clear the uterine lining after a miscarriage or abortion.
Ectopic Pregnancy The fertilized ovum implants in the fallopian tube and development occurs there, pregnancy cannot be sustained. Ampullary ectopic pregnancy is most common.
Causes of Ectopic Pregnancy Fallopian tube scarring or surgery prior, congenital tube defects, endometriosis, previous EP, exposure to DES (diethylstilbestrol-estrogen)
Symptoms of Ectopic Pregnancy Lower abdominal pain (one-sided, dull then sharp), delayed menses, abnormal vaginal bleeding, breast tenderness, nausea, positive pregnancy test, syncope, low Hct and Hgb, >12,000 WBC count
DX of Ectopic Pregnancy Pelvic Exam, Ultrasound (no uterine sac), B-hCG< 1500-2000. Serum progesterone <5 ng/dL, or serial serum B-hCG that is decreasing or at nonviable level.
Beta Human Chorionic Gonadotropin (B-hCG) EP is suspected if B-hCG is present at lower levels than expected for gestation. B-hCG peaks around week 8-10 of pregnancy.
TX of Ectopic Pregnancy Methotrexate if tube is not ruptured, pregnancy is 3.5 cm or less, no fetal heart movement, woman is stable. Follow up with B-hCG levels. OR Surgery: Salpingostomy *Give Rhogam for Rh- women
Methotrexate Used for TX ectopic pregnancy. MOA: inhibits cell division in embryo. SE: N/V, abdominal pain Educations: no alcohol or folic acids for a while after TX, monitor B-hCG levels, avoid intercourse till levels drop
Salpingostomy Surgery for treatment of EP. May be via laparoscopy. Fallopian tube is lacerated and maintained if viable, but may need to be removed.
Nursing Care for Ectopic Pregnancy Prevent hypovolemia, manage pain, support the woman and validate loss, screen high risk women, educate about safe sex practices to decrease incidence STI's, treat complications.
Gestational Trophoblastic Disease Abnormal proliferation of trophoblastic cells (outermost layer) of developing blastula
3 Types of GTD 1) Complete/Partial Hydatiform Mole 2) Invasive Mole 3)Choriocarcinoma
Molar Pregnancy Abnormal proliferation of placental tissue- chorionic villi and edema. Results in pregnancy loss, increased chances of choriocarcinoma.
Complete Hydatiform Mole Ovum develops with NO genetic material and becomes fertilized by sperm. Low chance of development of choriocarcinoma.
Partial Hydatiform Mole 69 Chromosomes in cell due to doubling of paternal chromosomes. Fetal tissue membranes are present. Can be extremely dangerous for mother, uterine rupture may occur.
S/S of Hydatiform Mole DARK brown spotting, profuse hemorrhage, uterus larger than expected, severe nausea, precclampsia prior to 24 weeks, greater than normal B-hCG, absent FHT's. US reveals grape-like clusters.
TX of Hydatiform Mole D&C. Follow with Chest X-ray, pelvic exam, B-hCG levels every 1-2 weeks x3 and every 1-2 months x 1 year. Avoid pregnancy!
Placenta Previa Placenta in the lower segment of the uterus, may cover cervical os. Unknown origin, occurs in about 1/200-300 pregnancies
Three types placenta previa 1)Total (complete) 2) partial 3) marginal (not covering cervix)
Risk Factors for Placenta Previa >35 years of age, smoking or Rx use, large placenta, previous C-section, close pregnancies
Management of Placenta Previa DX by ultrasound, C-section for complete placenta pre via, NO vaginal exams!!!!! (could lead to rupture of placenta). May labor if marginal. Frequent EFM, if non-reassuring need emergent C-section.
Symptoms of Placenta Previa Usually painless, FRANK red blood, fetal malpresentation (not engaged, relaxed, soft-nontender uterus
Nursing Intervention: Placenta Previa Monitor maternal VS frequently, document bleeding characteristics, pain if present, contractions (look for preterm labor), continuous EFM
Abruptio Placenta Placenta detaches from the uterine wall before fetus is born. Is a medical EMERGENCY! Occurs in 1/100 births, primary cause is unknown.
Risk factors for Abruptio Placenta Abdominal trauma, HTN, smoking, Cocaine abuse, History of previous abruption
Maternal Risks Associated with Placental Abruption Hemorrhage, shock, D&C, anemia, infection, hysterectomy, death
Fetal Risks after placental abruption HYPOXIA, brain damage, prematurity, anemia, Intrauterine growth restriction (IUGR) d/t inadequate nutrition, death
Most common symptoms of abruptio placenta Vaginal bleeding that's DARK red, dull aching pain in low back that can be SEVERE, uterine tenderness, decreased resting tone between contractions, rigid board-like abdomen, low intensity contractions
Nursing Assessment for Abruptio Placenta Maternal VS (oxygen, pain, and BP), EFM, abdominal assessment- resting tone between contractions, symptoms of shock, peri-pads to measure amount of blood,
Nursing Interventions for Abruptio Placenta IV start (>18 gauge for blood products), insert foley catheter, continuous EFM, oxygen administration (8-10L/min), prep for delivery or C-section, Type and Cross blood (at least 4 units!), measure abdominal girth, monitor DIC labs (fibrinogen, platelets, PT and aPTT, fibrin degradation product)
Disseminated Intravascular Coagulation (DIC) pathological process characterized by the widespread activation of the clotting cascade that results in the formation of blood clots in the small blood vessels throughout the body.
Uterine Inversion Cause of Intrapartum bleeding due to prolapse of the uterus during 3rd stage of labor (delivery of placenta) that occurs in 1/2000 births. Results in massive hemorrhage, maternal shock, and pain.
Risk factors for Uterine Inversion Pulling on the umbilical cord, fundal pressure during birth, increased abdominal pressure during birth, weak uterine wall and increased uterine relaxation, administration of Mg Sulfate
Uterine Rupture Tear in the uterine wall d/t increased uterine pressure during labor. Full rupture is rare but dehiscence is not.
Risk Factors for Uterine Rupture Previous C-section (increased risk with classic incision versus low-transverse), thin uterus wall, blunt trauma, and excessively strong contractions
S/S of Uterine Rupture May have no S/S. Pain between scapula (blood pools below diaphragm), a feeling that something "ripped", abdominal tenderness, chest/shoulder pain, hypovolemic shock, non-reassuring EFM, absent uterine contractions!
TX of Uterine Rupture Emergency operation to repair, emergent delivery of fetus, about 1/3 women require emergent hysterectomy
Postpartum Hemorrhage IS most common cause of maternal mortality/morbidity in the U.S.
Priorities during PPH 1) massage fundus 2) empty bladder 3) Administer oxytocic Rx's
Definition of PPH More than 500 mL blood loss after vaginal delivery or more than 1000 mL blood loss after C-section. *1g= 1mL S/S of shock don't appear until 1000-2000 mL have already been lost (30-40% total blood volume)
Symptoms of Hypovolemic Shock Shaking, pallor, tachycardia, increased Respiratory rate, restlessness and anxiety, cool, clammy skin, decreased blood pressure
Late PPH Begins later than 24 hours after birth and may continue for up to 6 to 12 weeks after. Usually not greater than 1 to 2 weeks after.
Causes of late PPH Sub-involution of uterus, infection with chlamydia, retained placental fragments
SX of Late PPH Lower abdominal tenderness, may have fever, lochia rubra beyond 1 week, foul discharge, fundal height greater than expected
TX of Late PPH Methergine 0.2 mg PO every 4 hours x 2days, antibiotics, D&C
Early Postpartum Hemorrhage Occurs within the first 24 hours after delivery.
Causes of early PPH UTERINE ATONY, genital trauma, and retained placental fragments. Hematoma, uterine inversion, and coagulation disorders.
Uterine Atony / Risk Factors for PPH Over-worked muscle from rapid or long lasting labor, use of oxytocin or uterine relaxing Rx's (Mg Sulfate), multiparous women (> 5 births), use of forceps or vacuum extraction, precclampsia
Recognition of PPH Identify risk factors early on, boggy enlarged uterus after delivery, large gush of blood, increased lochia flow (1 pad in 1 hr is bad!), clots, increased HR and RR, decreased BP, MAP, and urine output (are later signs!) Altered level of consciousness
Colloid IV Fluids Volume expanders, remain in the vascular space. They are expensive and need refrigeration. Examples: Dextrans and hetastarches
Crystalloid IV Fluids Don't stay in Intravascular space for long, used for hydration and calories. Examples: NS, LR, Dextrose (D5W). Are cheap and can be stored at room temperature.
Oxytocic Drugs Drugs used that have similar effects of oxytocin: promote uterine contraction and cease bleeding.
Oxytocin Given IM or IV, causes smooth muscle contraction.
Methergine NOT for use in Hypertensive clients. Given IM or PO. Causes uterine and vascular smooth muscle contraction
Hemabate Do not use for those with renal or liver impairments, glaucoma, asthma, or HTN. Injected into the uterus or given IM, causes myometrial contraction
Cytotec [Misopristol] POWERFUL myometrial contractions. Given rectally, not for use in those sensitive to prostaglandins.
Medical Intervention of PPH Bimanual compression, embolization of pelvic vessels, vessel ligation, or hysterectomy as last resort
Hematomas Cause of bleeding after delivery. Blood collects in connective or soft tissue under skin. Causes include trauma or injury to blood vessels, inadequate hemostasis at an incision or laceration site.
Risk Factors for Hematomas Episiotomies, laceration, nulliparity, operative vaginal delivery (forceps or vacuum), impaired coagulation
Nursing Intervention for Hematomas Frequently inspect site, observe for S/S of shock and report immediately, manage pain
TX of hematomas 3-5 cm hematomas only require palliative treatment such as Ice and analgesia. >5 cm hematomas may require incision, drainage, vessel ligation in the OR with sedation. ICE for 24 hours!
Lacerations Tear in the perineum, vagina, or cervix. Can include 1 large laceration or several small lacerations.
Risk factors for Laceration Younger maternal age, nulliparity, epidural anesthesia, fast childbirth, forceps or vacuum delivery, macrosomia, use of oxytocin
SX of Lacerations PERSISTENT BLEEDING DESPITE FIRM FUNDUS, symptoms of hypovolemic shock, pain
Nursing Management of Lacerations Notify MD ro CNM immediately. May require immediate repair to stop bleeding.
Ongoing care for PPH I/O, IV access with > 18 gauge, rest, pain management, help care for baby, Iron supplement with vitamin C, education, communication with health staff, and report symptoms of infection
Show full summary Hide full summary

Similar

Nervous System
4everlakena
Diabetes Mellitus
Kirsty Jayne Buckley
Renal System A&P
Kirsty Jayne Buckley
Oxygenation
Jessdwill
Clostridium Difficile
Kirsty Jayne Buckley
Definitions
katherinethelma
Clinical Governance
Kirsty Jayne Buckley
CMS Interpretive Guidelines for Complaint/Grievances
Lydia Elliott, Ed.D
NCLEX RN SAMPLE TEST
MrPRCA
NURS 310 EXAM 1 PRACTIC EXAM
harlacherha
Skin Integrity and Wound Care
cpeters