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15617047
Acute Myocardial Infarction
Description
Mind Map on Acute Myocardial Infarction, created by Jaimee Plumley on 17-10-2018.
Mind Map by
Jaimee Plumley
, updated more than 1 year ago
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Created by
Jaimee Plumley
about 6 years ago
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Resource summary
Acute Myocardial Infarction
Assessment
clinical manifestations
subjective
Nausea - SNS Activation
Anxiety
SOB, Dyspnea or Orthopnea
Chest pain - Crushing, heaviness, pressure
Pain may radiate to neck, jaw, back and arms
Objective
Fever - inflammatory response
Vitals
High BP and High HR
Last stages decrease BP d/t decrease CO
Sweating - SNS activation
Syncope / Presyncope
Skin - cool clammy bc SNS
Crackles in lungs left ventricular ischemia
Jugular vein distention, peripheral edema right ventricular ischemia
Abnormal Heart Sounds - S3 or S4 - indication of ventricular dysfunction
Hyperkalemia - Acidosis - Necrosis
S/S difference from Men and Women
risk factors
non-modifiable
African Amerian Decent
Family History
Age Men > 45 & Women > 55
CAD - Coronary Artery Disease
modifiable
Smoking
obesity
stress
high LDL & low HDL
lack of physical activity
hypertension
Treatment
pharmaceutticals
Intravenous Nitroglycerin
goals of therapy
reduce angina pain
improve coronary blood flow
effects
preload
afterload
oxygen supply
adverse effects
hypotension
monitor BP
tolerance
administer a lower dose at night and a higher dose during the day
Morphine Sulphate
used for chest pain not relieved by nitroglycerin
effects (vasodilator)
Cardiac Workload by
myocardial oxygen consumption
contractility
BP
HR
adverse effects
respiratory depression
monitor for signs of bradypnea and hypoxia
B-Adrenergic Blockers
NOT given by IV to patients with STEMI
S-T Elevation Myocardial Infarction
administered PO within 24 hours of STEMI
effects
myocardial oxygen demand
HR
BP
Contractility
Angiotensin-Converting Enzyme Inhibitors
recommended
Anterior wall MI
Infarctions that cause decreased left ventricular function
pulmonary congestion
Antidysrhythmia Medications
Cholesterol- Lowering Drugs
Stool Softeners
Post MI patients are on bedrest and are administered opioids
increases risk of constipation
Colace
prevents straining and vagal stimulation
non-pharmeceuticals
Nutritional
NPO (pt may sip water) until stable
Coronary Surgical Revascularization
Coronary Artery Bypass Graft Surgery (CABG)
Construction of new conduits between major arteries and the myocardium distal to the obstructed coronary arteries
needs
sternotomy
opening of the chest cavity
cardiopulmonary bypass (CPB)
diverts patient's blood from the heart to the CPB machine
the CPB machine oxygenates the blood
blood is pumped back into patient
Minimally Invasive Direct Coronary Artery Bypass
for patients with single vessel disease
left anterior descending
Right sided Coronary Artery Disease
does not involve a sternotomy and CPB
Emergent Care
Administer Oxygen
Nitrates for Chest Pain
Opioid analgesic
Vitals
Telemetry
White is Right, Black is Opposite, Snow(White) over Trees(Green), Smoke(Black) over Fire(Red)
Heart Sounds
Comfortable Positon
PCI (percutaneous coronary intervention)
catheter equipped with an inflatable balloon tip is inserted into a narrowed coronary artery and the balloon is inflated
Diagnosis
Diagnostic Testing
ECG
help identify areas of your heart that are dysfunctioning
STEMI (ST elevation myocardial infarction) with pathological Q waves
Bloodwork
check for proteins that are associated with heart damage, such as serum cardiac markers
Specific to MI
Cardiac specific tropinin T (cTnT)
Cardiac specific tropinin I (cTnI)
Creatine Kinase muscle brain (CK-MB) levels rise 3 - 12 hrs after MI
indication of myocardial damage
Myoglobin is a small, oxygen-binding protein found in heart and skeletal muscle
Early MI indication - it is release in the blood after heart injury
Stress Test
see how your heart responds to certain situations, such as exercise
Angiogram
coronary catheterization to look for areas of blockage in your arteries
help indicate a need for PCI (percutaneous Coronary Intervention)
Special Consideration
Women are often misdiagnosised
MI’s in women are usually more fatal than in men because:
symptoms in females are less typical (less severe chest pain, often reported as severe heartburn or pain in the breast)
do not seek treatment early in attack or at all
onset is later (>65 yrs) when other health conditions may contribute or complicate
smaller coronary arteries
Elderly are also misdiagnosis
Decrease CO leads to decrease LOC
decrease LOC is mistaken for delirium
Pathophysiology
Irreversible myocardial cell death due to lack of blood flow
Zones
1. Necrotic zone 2. Injured area: some cells may recover 3. Ischemic zone: cells will recover if perfusion is restored
Caused usually by thrombus leading to distal portions of the heart to necrosis
Heart is able to withstand 20 mins of ischemic condition then cell death begins
Starting from the inner layer to the outer it takes about 5-6 hours to become full necrotic in an area
Necrosis can happen anywhere in the heart and it correlates to the circulation that is impaired
For ex: Anterior wall infractions come from the right coronary artery occlusion
METAPHOR
Think of a one way road and an accident stopping traffic
Accident is Thrombus
Cars are Blood flow
Complications
Dysrhythmias
Most Common complication
Ventricular fibrillation
Occurs within 4 hours of onset of pain
Premature Ventricular Contractions
May precede ventricular tachycardia and fibrillation
Heart Failure
Pumping power of heart has decreased
Manifestations
Dyspnea, restlessness, agitation, tacycardia
S4 and S3 sounds, crackles, distended jugular veins
Cardiogenic Shock
Inadequate oxygen and nutrient supply to tissues
Papillary Muscle dysfunction
Occurs if infarcts area includes papillary muscles that attaches to mitral valve
Causes mitral valve regurgitation
Increases volume of blood in left atriu
Decreases CO
Can lead to papillary rupture (rare)
Results in dyspnea, pulmonary edema, and decreased CO
Patient’s condition decreases rapidly
Ventricular Aneurysm
occurs when infarcted myocardial wall thins and bulges during contraction
refractory heart failure, angina,
dysrhythmias
can lead to embolic stroke
can lead to
Pericarditis
inflammation
visceral
parietal pericardium
chest pain (may be relived by sitting), coughing,
Dressler's Syndrome
pericarditis + effusion and fever that develops 4-6 weeks after MI
potentially occurs after open heart surgery
manifestations
pericardial pain
fever
friction rub
plural effusion
arthralgia
Nursing Process
Planning
Care Goals
relief of pain
preservation of myocardium
treatment of ischemia
coping with anxiety
patient participation in rehabilitation plan
reduction of risk factors
Implementation
Patient Education
Physical Activity
5 or more times per week
intensity is determined by HR
HR during activity should not be more than 20 beats/min over resting HR
type
regular, rhythmic, and repetitive; large muscles should be used
Walking
cycling
swimming
rowing
Gradual resumption of activities
Activity should not be abruptly started or stopped
stretch for 3-5 minutes before and after activity
S/S of angina and Acute MI
why angina and MI occur
When and how to seek help
Definition of terms
CAD
Acute MI
HF
Healing after an MI
promote recovery
decrease Risk Factors
Sexual activity
Wait 3-4 hours after a large meal before sexual activity
Hot or cold showers should be avoided before and after
Erectile agents are contradicted if patient is taking nitrates
Prophylactic use of nitrates decreases angina during sexual activity
Anal intercourse may cause cardiac stress
Can Induce vasovagal response
Nursing Diagnosis
Acute Pain
Evaluate PQRST and alleviating factors
monitor
cardiac rhythm and rate
BP
hemodynamic parameters
effectiveness of O2 therapy
administer pain medication
Obtain 12-lead ECG during pain episode
Decreased Cardiac Output
monitor
Vital signs
dysrhythmias
Respiratory
watch for signs of HF
Fluid balance
observe for fluid retention
Arrange exercise and periods of rest
Anxiety
observe for S/S
Assess for changes in anxiety
anxiety increases O2 needs
teach relaxation techniques
deep, relaxed breathing
Encourage patient to voice fears and feelings
Activity Intolerance
monitor response to antidysrhythmic medications before activity
arrange for exercise and rest periods
teach patient about energy conservation
teach patients pacing techniques for ADL's and to self monitor O2
Evaluation
Expected Outcomes
understands medication
purpose
action
dosage
route
leads to compliance of medication
pain relief and optimal conditions
Health Teaching
Understands the disease process
able to explain acute MI in their own words
anxiety relief
lifestyle changes
reducing risk factors
Avaliblity to local community resources
Reports Relief Of pain
Stable signs of effective cardiac perfusion
Able to tolerate activity
Nursing Assessment
Refer to Red Assessment Bubble for S/S
Head to Toe Assessment
Priority
Cardiac
Respiratory
Neurological
Past Medical History
Current Medical History
Current Medication
Jaimee Plumley 215 149 784 & Vaanathy Sivanandan (214 237 929)
Media attachments
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Heartfailure (binary/octet-stream)
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