7033462
Description
Mind Map by ahmed king, updated more than 1 year ago
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Created by ahmed king
about 8 years ago
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|
Burns
- types
- Heat burns
- Cold temperature burns
- Electrical burns
- Chemical
burns
- Radiation
burns
- Friction burns
- Heat burns
- skin
- epidermis
- stratum corneum
- sratum granulosum
- stratum spinosm
- stratum basale
- stratum lucedium
- stratum corneum
- dermis
- Appendages
- Sebaceous glands (sebum)
- Empty into hair follicle
- Directly into
skin surface
- Empty into hair follicle
- Sweat glands
- eccrine
- apocrine
- eccrine
- Hair and hair follicles
- arrector pili
- arrector pili
- Sebaceous glands (sebum)
- Appendages
- hypodermis
- adipose tissue
- adipose tissue
- epidermis
- treatment
- onitment
- topical therapy
- providing occlusion,
hydration, and lubrication.
- topical therapy
- Cooling
- Pain management
- Dressings
- onitment
- Classification
- First-degree
- affect only the epidermis
- affect only the epidermis
- second degree
- epidermis and part of
the dermis
- epidermis and part of
the dermis
- third degree
- epidermis and dermis and may go
into the subcutaneous tissue
- epidermis and dermis and may go
into the subcutaneous tissue
- Fourth
degree
- damage the underlying bones,
muscles, and tendons
- damage the underlying bones,
muscles, and tendons
- First-degree
- antibiotics
- Mechanism & types
- Mycolic acid
synthesis
inhibitors
- folic acid
synthesis
inhibitors
- cell wall
synthesis
inhibitors
- Penicillin
- Types
- Penicillins
- gram-positiveorganisms
- gram-negative cocci
- andnon-β-lactamaseproducinganaerobes
- Susceptible to
hydrolysis by
β-lactamases
- gram-positiveorganisms
- Antistaphylococcal Penicillins
- Resistant to
staphylococcal
β-lactamases
- staphylococci
- streptococci
- Resistant to
staphylococcal
β-lactamases
- Extended-SpectrumPenicillins
- gram-negativeorganisms
- Susceptible
to hydrolysis
by
β-lactamases
- gram-negativeorganisms
- Penicillins
- Mechanism
- bind covalently to
PBP and inhibit the
transpeptidation
process leading to
cell death
- bind covalently to
PBP and inhibit the
transpeptidation
process leading to
cell death
- Pharmacokinetics
- Stable for years in dry form
but loses activity rapidly (24
hrs at 20°C) in solution
form.
- Elimination
- renal excretion
- active
tubular
secretion
- active
tubular
secretion
- renal excretion
- Absorption
- Impaired by food
- Most tissue
concentrations are
equivalent to those
in serum
- Impaired by food
- Stable for years in dry form
but loses activity rapidly (24
hrs at 20°C) in solution
form.
- Types
- Penicillin
- inhibition of
protein synthesis
- RNA synthesis
inhibitors
- DNA synthesis
inhibitors
- Mycolic acid
synthesis
inhibitors
- Mechanism & types
- Burn center
- Al Wakra Hospital
- Al Wakra Hospital
- Microbiology lab
- testing, culturing, and
research
- study
micro-organisms
- study
micro-organisms
- testing, culturing, and
research
- cell death
- necrosis
- Pathologic death of one or
more cells, or of a portion of
tissue or organ, resulting
from irreversible damage
- Pathologic death of one or
more cells, or of a portion of
tissue or organ, resulting
from irreversible damage
- apoptosis
- programmable
cell
death
- programmable
cell
death
- necrosis
- inflamation
- cardinal signs
- Pain
- Redness
- Immobility
- Swelling
- Heat
- Pain
- steps
- recognizing os
the forien
bodies by
host cells and
molecules
- recruitment
of the
Leukocytes
and plasma
proteins
- leukocytes
and
proteins
are
activated
to
eliminate
the
offending
substance
- damaged
tissue
is
repaired
- recognizing os
the forien
bodies by
host cells and
molecules
- cardinal signs
- infection
- Lymph Nodes
- Functions
- Removal of
foreign
materials such
as bacteria and
tumor cells from
the lymphatic
stream
- Production of
lymphocytes
that function in
the specific
immune
response
- Removal of
foreign
materials such
as bacteria and
tumor cells from
the lymphatic
stream
- Lymphadenitis
- enlarged by swelling
(inflammation), often in
response to bacteria,
viruses, or fungi
- Treatment
- Antibiotics to
treat bacterial
infections
- Analgesics
(painkillers)
to
control
pain
- Anti-inflammatory
medicines
to
reduce
inflammation
- Antibiotics to
treat bacterial
infections
- enlarged by swelling
(inflammation), often in
response to bacteria,
viruses, or fungi
- Functions
- Immunity
- Innate
- protects the
body from
any
pathogens.
- protects the
body from
any
pathogens.
- Adaptive
- acts against specific
foreign substances.
- acts against specific
foreign substances.
- Innate
- Lymph Nodes
- healing
- phases
- Inflammatory
- Hemostasis
- 1-Vasoconstriction
2-Platelet plug
formation
3-Coagulation
- 1-Vasoconstriction
2-Platelet plug
formation
3-Coagulation
- Hemostasis
- Proliferative
- Epithelization
- basal cell proliferation
and epithelial migration
occurring in the fibrin
bridgework inside a clot.
- basal cell proliferation
and epithelial migration
occurring in the fibrin
bridgework inside a clot.
- Fibroplasia
- fibroblast
proliferation,
accumulation of
ground substance,
and collagen
production
- fibroblast
proliferation,
accumulation of
ground substance,
and collagen
production
- Angiogenesis
- formation of
new blood
vessels
- formation of
new blood
vessels
- Epithelization
- Maturational
- collagen cross-linking,
collagen remodeling,
wound contraction and
repigmentation
- collagen cross-linking,
collagen remodeling,
wound contraction and
repigmentation
- Inflammatory
- Classification
of Wounds
Closure
- Primary
Intention
- All
Layers
are
closed
- All
Layers
are
closed
- Secondary
Intention
- Deep layers are closed
but superficial layers
are left to heal from
the inside out
- Deep layers are closed
but superficial layers
are left to heal from
the inside out
- Tertiary
Intention
- delayed
primary
closure
- delayed
primary
closure
- Primary
Intention
- Risk
factors for
non
healing
- Aging
- Infection
- Diabetes
- Nutritional
status
- Smoking
- Aging
- phases
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