Zusammenfassung der Ressource
Cardiac Arrhythmias
- Supraventricular
arrhythmias
- Atrial Fibrillation
- Ax: Cardiac : IHD / Valve
disease / HTN Metabolic:
Thyrotoxicosis, alcohol
binge, electrolyte abnormalities
- Sx: Palpitations /
syncope / SOB
- Dx: ECG - absent P
waves / O/E - irregularly
irregular pulse
- Management
- Acute AF
- <48 hours: 1. Rate control 2.
TOE 3. No thrombus -> DC /
chemical cardioversion NB: if
CV score is >=2 then Heparin at
the same time.
- Haemodynamically
Unstable: Immediate
DC cardioversion
- Chemical
cardioversion -
flecainide /
amiodarone
- >48 hours: 1. Rate control 2. TOE
3. No thrombus then heparin
until ApTT 45-60 then cardiovert
NB: if CV score >=2 then anticoag
for 3-4 weeks first
- TOE: Evidence of thrombus -
anticoag for 3-4 weeks then
cardiovert
- Chronic AF
- Rate control (elderly /
permenant AF) - B-blockers
and Non-DHP CCB. Heart
failure? - use digoxin and
amiodarone
- Rhythm control (young /
paroxysmal / persistent AF /
symptomatic) - Flecainide /
amiodarone then catheter
ablation
- CHADSVASC score
- Need for
anticoagulation -
post cardioversion
needed for up to 4
weeks
- Atrial Flutter
- Supraventricular atrial
tachycardia. Re-entrant cycle
set up - 300bpm but normally
2:1 block from AVN
- Sx: Syncope /
palpitations /
SOB
- Dx: ECG - saw tooth
pattern. Carotid sinus /
adenosine to help
diagnosis
- Ax: Atrial
abnormalities incl. AF
/ thyrotoxicosis /
alcoholism / chronic
lung conditions
- Treatment
- Rate control -
B-blockers and non-DHP CCBs
- Rhythm control - DC
synchronised cardioversion
(50J) / Catheter ablation
- Needs anticoagulation
beforehand / After? -
CHADSVASC
- AV Node
re-entrant
tachycardia
- Re-entrant cycle
set up within AVN
- Tx: Vasotonic manoeuvres /
break re-entrant cycle - IV
adenosine / DC synchronised
cardioversion (50J)
- Accessory pathway
re-entrant
tachycardia
- WPW syndrome - accessory
pathway that allows
impulse to reach ventricles
faster
- Sx: Sudden
palpitations /
syncope / SOB
- WPW + AF - extremely
dangerous and can cause
VF - DC cardioversion
- Dx: ECG - delta wave
and short PR
interval
- Long term tx: Rate
control - BBs and CCBs
/ catheter ablation
- Ventricular
arrhythmias
- Ventricular Fibrillation
(VF)
- Rapid ventricular
rhythm that doesn't
allow ventricles to
contract properly
- Causes - IHD / R on
T (ventricular
ectopics) / long QT
- Monomorphic /
polymorphic
(Torsades)
- ALS Algorithm - shockable rhythm
- Ventricular
Tachycardia
- Ventricular
rhythm
>100bpm
- Ax: Re-entry loop -
damaged heart / triggered
automaticity - damaged
heart / digoxin
- ALS guidelines 1) Not
stable - shockable 2)
Stable - ABCDE and
correct causes
- Ventricular
Ectopics
- Ax: Idopathic - harmless in
those with normal hearts Precipitant -
caffeine / alcohol / digoxin | Post-MI /
heart damage / digoxin
- Harmless ectopics often
disappear with exercise
- Dx: ECG - premature
ventricular beats (can
appear as bigeminy /
trigeminy
- Sx: Skipped beat feeling
- Tx: Nothing / remove
precipitants / rate control
with B-blockers if
symptomatic
- Anti-arrhythmics
NOT used -
increase risk of
death
- Atrioventricular
Block
- First degree block:
PR >0.2 seconds
- Physiological
- Second
degree block
- Mobitz I: Wenkebach
phenomenon -
increasing PR till drop
of QRS complex
- Ax: May by physiological /
underlying heart condition
- Mobitz II: PR interval
constant but
intermittent loss of QRS
complex (2:1 / 3:1 block)
- Ax: Always pathological -
damage to conducting
fibres e.g. post MI
- Pacemaker
required
- Third degree block -
Complete block
- Complete dissociation
between P waves and
QRS complexes
- Tx: Transient - Atropine /
stop Digoxin. Permenant -
Pacemaker needed
- Ax: May be transient
post- MI (atropine) /
permenant - Inferior MI
(damage to AVN)
- Sx: Depends on location
of escape beat 1. Above
bundle = mild Sx and
narrow QRS 2. In bundle =
severe Sx and wide QRS
- Bundle branch
block
- RBBB
- Ax: Physiological /
new-onset may be anterior MI
related
- Dx: ECG - M sign in
right sided leads
(V1/V2) / wide QRS
- LBBB
- Ax: Always
pathological e.g MI
/ HTN / valve
disease
- Dx: ECG - M sign in
left sided ant.
leads (V5/V6) / wide QRS
- Hemifasicular
block
- LBB divides into anterior
and posterior
hemifasciles
- Damage causes
change in axis
- Drug
toxicity
- Digoxin
- Normal action: Reduce heart rate
(slows AV transmission) / increases
contractility by decreasing
repolarisation time
- Used for patients with HF
to reduce oedema
- Works by inhibiting
the NaKATPase
pump
- Toxicity
- Ax: Overdose/ Renal impairment
/ electrolyte abnormalities -
hypokalaemia / low Mg / Drugs -
CCBs / amiodarone
- Sx: Confusion / weakness /
syncope / palpitations / N&V
and abdo pain / yellow haze
- ECG: Increase PR / PVCs /
scooped ST segment
- Long QT
syndrome
- Ax: Congenital - genetic
mutation in Na+ / K+
channels. Acquired -
hypocalcaemia /
hypokalaemia / low Mg /
SAH
- Dx: ECG -
long QTc / U&Es
- Sx: Cardiac
arrest /
syncope
- Torsades de pointes -
polymorphic VT that can go
into VF / treat with
Magnesium sulphate and
defib
- Tx: Remove or
correct
precipitants,
ICD