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11845576
Cardiac Arrhythmias
Descripción
Overview of the common cardiac arrhythmias
Sin etiquetas
arrhythmias
cardiac
medicine
Mapa Mental por
Louis Darby
, actualizado hace más de 1 año
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Menos
Creado por
Louis Darby
hace casi 7 años
154
1
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Resumen del Recurso
Cardiac Arrhythmias
Supraventricular arrhythmias
Atrial Fibrillation
Ax: Cardiac : IHD / Valve disease / HTN Metabolic: Thyrotoxicosis, alcohol binge, electrolyte abnormalities
Sx: Palpitations / syncope / SOB
Dx: ECG - absent P waves / O/E - irregularly irregular pulse
Management
Acute AF
<48 hours: 1. Rate control 2. TOE 3. No thrombus -> DC / chemical cardioversion NB: if CV score is >=2 then Heparin at the same time.
Haemodynamically Unstable: Immediate DC cardioversion
Chemical cardioversion - flecainide / amiodarone
>48 hours: 1. Rate control 2. TOE 3. No thrombus then heparin until ApTT 45-60 then cardiovert NB: if CV score >=2 then anticoag for 3-4 weeks first
TOE: Evidence of thrombus - anticoag for 3-4 weeks then cardiovert
Chronic AF
Rate control (elderly / permenant AF) - B-blockers and Non-DHP CCB. Heart failure? - use digoxin and amiodarone
Rhythm control (young / paroxysmal / persistent AF / symptomatic) - Flecainide / amiodarone then catheter ablation
CHADSVASC score
Need for anticoagulation - post cardioversion needed for up to 4 weeks
Atrial Flutter
Supraventricular atrial tachycardia. Re-entrant cycle set up - 300bpm but normally 2:1 block from AVN
Sx: Syncope / palpitations / SOB
Dx: ECG - saw tooth pattern. Carotid sinus / adenosine to help diagnosis
Ax: Atrial abnormalities incl. AF / thyrotoxicosis / alcoholism / chronic lung conditions
Treatment
Rate control - B-blockers and non-DHP CCBs
Rhythm control - DC synchronised cardioversion (50J) / Catheter ablation
Needs anticoagulation beforehand / After? - CHADSVASC
AV Node re-entrant tachycardia
Re-entrant cycle set up within AVN
Tx: Vasotonic manoeuvres / break re-entrant cycle - IV adenosine / DC synchronised cardioversion (50J)
Accessory pathway re-entrant tachycardia
WPW syndrome - accessory pathway that allows impulse to reach ventricles faster
Sx: Sudden palpitations / syncope / SOB
WPW + AF - extremely dangerous and can cause VF - DC cardioversion
Dx: ECG - delta wave and short PR interval
Long term tx: Rate control - BBs and CCBs / catheter ablation
Ventricular arrhythmias
Ventricular Fibrillation (VF)
Rapid ventricular rhythm that doesn't allow ventricles to contract properly
Causes - IHD / R on T (ventricular ectopics) / long QT
Monomorphic / polymorphic (Torsades)
ALS Algorithm - shockable rhythm
Ventricular Tachycardia
Ventricular rhythm >100bpm
Ax: Re-entry loop - damaged heart / triggered automaticity - damaged heart / digoxin
ALS guidelines 1) Not stable - shockable 2) Stable - ABCDE and correct causes
Ventricular Ectopics
Ax: Idopathic - harmless in those with normal hearts Precipitant - caffeine / alcohol / digoxin | Post-MI / heart damage / digoxin
Harmless ectopics often disappear with exercise
Dx: ECG - premature ventricular beats (can appear as bigeminy / trigeminy
Sx: Skipped beat feeling
Tx: Nothing / remove precipitants / rate control with B-blockers if symptomatic
Anti-arrhythmics NOT used - increase risk of death
Atrioventricular Block
First degree block: PR >0.2 seconds
Physiological
Second degree block
Mobitz I: Wenkebach phenomenon - increasing PR till drop of QRS complex
Ax: May by physiological / underlying heart condition
Mobitz II: PR interval constant but intermittent loss of QRS complex (2:1 / 3:1 block)
Ax: Always pathological - damage to conducting fibres e.g. post MI
Pacemaker required
Third degree block - Complete block
Complete dissociation between P waves and QRS complexes
Tx: Transient - Atropine / stop Digoxin. Permenant - Pacemaker needed
Ax: May be transient post- MI (atropine) / permenant - Inferior MI (damage to AVN)
Sx: Depends on location of escape beat 1. Above bundle = mild Sx and narrow QRS 2. In bundle = severe Sx and wide QRS
Bundle branch block
RBBB
Ax: Physiological / new-onset may be anterior MI related
Dx: ECG - M sign in right sided leads (V1/V2) / wide QRS
LBBB
Ax: Always pathological e.g MI / HTN / valve disease
Dx: ECG - M sign in left sided ant. leads (V5/V6) / wide QRS
Hemifasicular block
LBB divides into anterior and posterior hemifasciles
Damage causes change in axis
Drug toxicity
Digoxin
Normal action: Reduce heart rate (slows AV transmission) / increases contractility by decreasing repolarisation time
Used for patients with HF to reduce oedema
Works by inhibiting the NaKATPase pump
Toxicity
Ax: Overdose/ Renal impairment / electrolyte abnormalities - hypokalaemia / low Mg / Drugs - CCBs / amiodarone
Sx: Confusion / weakness / syncope / palpitations / N&V and abdo pain / yellow haze
ECG: Increase PR / PVCs / scooped ST segment
Long QT syndrome
Ax: Congenital - genetic mutation in Na+ / K+ channels. Acquired - hypocalcaemia / hypokalaemia / low Mg / SAH
Dx: ECG - long QTc / U&Es
Sx: Cardiac arrest / syncope
Torsades de pointes - polymorphic VT that can go into VF / treat with Magnesium sulphate and defib
Tx: Remove or correct precipitants, ICD
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