Frontal Lobes Flashcards

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PSYB65 Fichas sobre Frontal Lobes Flashcards, creado por andreaarose el 17/12/2013.
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Anatomy of the frontal lobes Constitutes of 20% of the neocortex.
Subdivisions of the frontal lobe Motor and premotor areas.
Subdivisions of premotor areas Lateral area 6, medial area 6, area 8, area 8A.
Prefrontal cortex Area of the frontal lobe that receives input from the dorsomedial nucleus of the thalamus
Divisions of the prefrontal cortex Dorsolateral prefrontal cortex, inferior frontal cortex, medial frontal cortex.
Motor cortex Projects to spinal motor neurons, cranial nerves that control the face, projects to the basal ganglia and the red nucleus
Connections of the premotor areas Projects to the spinal cord and motor cortex. Receives projections from parietal areas PE and PF and dorsolateral prefrontal area.
Eye fields Receive from PG and the superior colliculus
Dorsolateral prefrontal area Reciprocal connections with the posterior parietal and STS, extensive connections with the cingulate cortex, basal ganglia, and superior colliculus, receives input from dopaminergic cells in tegmentum
Orbital frontal cortex Receives from the temporal lobe, amygdala, gustatory cortex, somatosensory cortex, olfactory cortex, dopaminergic cells in tegmentum, projects to hypothalamus and amygdala
Frontal lobe functions Planning and selection, persistence and ignoring distracting stimuli, memory for what you have already done, executive functions
Functions of the premotor cortex Selects movements to be executed, chooses behaviour in response to external cues.
Premotor cortex and movement An increase in activity in the premotor cortex is seen when cues become associated with movement
Functions of the prefrontal cortex An increase in activity in the premotor cortex is seen when cues become associated with movement
Internal cues Temporal memory - memory for what has just happened
External cues Feedback about rewarding properties of stimuli
Orbital frontal cortex Learning by association - external cues.
Context cues Orbital frontal cortex helps with context cues in social interactions.
Autonoetic awareness Self knowledge
Left frontal lobe Language, encoding memories.
Right frontal lobe Nonverbal movements, facial expression, retrieving memories.
Damage to the primary motor cortex Loss of fine movements, speed and strength.
Damage to the premotor or dorsolateral cortex Loss of movement programming.
Damage to the frontal eye fields Changes in voluntary gaze.
Damage to Broca's area Agrammatism
Damage to the supplementary motor cortex Become mute.
Convergent thinking Only one answer to the question
Divergent thinking Questions that ask for a variety of responses - frontal lobe patients are impaired.
Loss of behavioural spontaneity Decrease verbal fluency and design fluency and reduction in general behaviours in frontal lobe patients.
Frontal lobe lesions Increased perseveration, inability to form a strategy and loss of response inhibition, deficits in self regulation.
Loss of response inhibition Tested through the Wisconsin card sorting task and the Stroop test.
Risks Frontal lobe lesions - Iowa gambling taks, appears after damage to the orbitofrontal cortex.
Loss of associative learning Inability to select from competing responses
Pseudodepression Appears after lesions of the left frontal lobe, outward apathy, indifference, loss of initiative, reduced sexual interest, little or no verbal output
Pseudopsychopathy Appears after lesions of the right frontal lobe, immature, lack of tact and restraint, promiscuous coarse language, lack of social graces, increased motor activity
Orbitofrontal lesions Reduce inhibitions and may introduce abnormal sexual behavior, leads to deficits in identifying facial expressions
Dorsolateral lesions Reduces interest in sexual behaviour.
Damage to the frontal facial area Sensory and motor functions of the face are preserved after damage. Left = loss of verbal fluency, right = loss of design fluency
Schizophrenia Abnormality in the mesocortical dopaminergic projection. Decrease in blood flow to the frontal lobes, and frontal lobe atrophy
Parkinson's disease Loss of dopamine cells in the substantia nigra that project to the prefrontal cortex
Korsakoff's Alcohol-induced damage to the dorsomedial thalamus and a deficiency in frontal lobe catecholamines
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