Creado por andreaarose
hace casi 11 años
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Pregunta | Respuesta |
Anatomy of the frontal lobes | Constitutes of 20% of the neocortex. |
Subdivisions of the frontal lobe | Motor and premotor areas. |
Subdivisions of premotor areas | Lateral area 6, medial area 6, area 8, area 8A. |
Prefrontal cortex | Area of the frontal lobe that receives input from the dorsomedial nucleus of the thalamus |
Divisions of the prefrontal cortex | Dorsolateral prefrontal cortex, inferior frontal cortex, medial frontal cortex. |
Motor cortex | Projects to spinal motor neurons, cranial nerves that control the face, projects to the basal ganglia and the red nucleus |
Connections of the premotor areas | Projects to the spinal cord and motor cortex. Receives projections from parietal areas PE and PF and dorsolateral prefrontal area. |
Eye fields | Receive from PG and the superior colliculus |
Dorsolateral prefrontal area | Reciprocal connections with the posterior parietal and STS, extensive connections with the cingulate cortex, basal ganglia, and superior colliculus, receives input from dopaminergic cells in tegmentum |
Orbital frontal cortex | Receives from the temporal lobe, amygdala, gustatory cortex, somatosensory cortex, olfactory cortex, dopaminergic cells in tegmentum, projects to hypothalamus and amygdala |
Frontal lobe functions | Planning and selection, persistence and ignoring distracting stimuli, memory for what you have already done, executive functions |
Functions of the premotor cortex | Selects movements to be executed, chooses behaviour in response to external cues. |
Premotor cortex and movement | An increase in activity in the premotor cortex is seen when cues become associated with movement |
Functions of the prefrontal cortex | An increase in activity in the premotor cortex is seen when cues become associated with movement |
Internal cues | Temporal memory - memory for what has just happened |
External cues | Feedback about rewarding properties of stimuli |
Orbital frontal cortex | Learning by association - external cues. |
Context cues | Orbital frontal cortex helps with context cues in social interactions. |
Autonoetic awareness | Self knowledge |
Left frontal lobe | Language, encoding memories. |
Right frontal lobe | Nonverbal movements, facial expression, retrieving memories. |
Damage to the primary motor cortex | Loss of fine movements, speed and strength. |
Damage to the premotor or dorsolateral cortex | Loss of movement programming. |
Damage to the frontal eye fields | Changes in voluntary gaze. |
Damage to Broca's area | Agrammatism |
Damage to the supplementary motor cortex | Become mute. |
Convergent thinking | Only one answer to the question |
Divergent thinking | Questions that ask for a variety of responses - frontal lobe patients are impaired. |
Loss of behavioural spontaneity | Decrease verbal fluency and design fluency and reduction in general behaviours in frontal lobe patients. |
Frontal lobe lesions | Increased perseveration, inability to form a strategy and loss of response inhibition, deficits in self regulation. |
Loss of response inhibition | Tested through the Wisconsin card sorting task and the Stroop test. |
Risks | Frontal lobe lesions - Iowa gambling taks, appears after damage to the orbitofrontal cortex. |
Loss of associative learning | Inability to select from competing responses |
Pseudodepression | Appears after lesions of the left frontal lobe, outward apathy, indifference, loss of initiative, reduced sexual interest, little or no verbal output |
Pseudopsychopathy | Appears after lesions of the right frontal lobe, immature, lack of tact and restraint, promiscuous coarse language, lack of social graces, increased motor activity |
Orbitofrontal lesions | Reduce inhibitions and may introduce abnormal sexual behavior, leads to deficits in identifying facial expressions |
Dorsolateral lesions | Reduces interest in sexual behaviour. |
Damage to the frontal facial area | Sensory and motor functions of the face are preserved after damage. Left = loss of verbal fluency, right = loss of design fluency |
Schizophrenia | Abnormality in the mesocortical dopaminergic projection. Decrease in blood flow to the frontal lobes, and frontal lobe atrophy |
Parkinson's disease | Loss of dopamine cells in the substantia nigra that project to the prefrontal cortex |
Korsakoff's | Alcohol-induced damage to the dorsomedial thalamus and a deficiency in frontal lobe catecholamines |
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