Large animal plants

It is fall in VA, and a cattle farmer calls you concerned because one of his cows is having a seizure, and two others began acting strangely yesterday. He has not made any recent changes to their feed, but they are free to roam his 20 acres of pasture. You go over to investigate, and find the two of the affected cows to have a nystagmus and all of them are exhibiting a hypermetric gate (goose-stepping). You take a look around the property and notice a tree with yellow flowers, palmately compound leaves and large, brown seeds that one of the cows is showing interest in. Based on this, and the clinical signs, you conclude that this must be:

How might you treat the cows you saw at this farm?

Which of the following species are generally resistant to oak toxicity?

Which toxic trees are correctly matched with their toxic compound?

Which of these toxic trees can lead to laminitis?

It is spring in Oregon and you are called out to examine some cattle that have fallen sick over the course of about a week. Some have black tarry stool, a few have ventral edema and nasal discharge and all of them seem lethargic. The farmer reports that one of the cows seemed very thirsty yesterday, and died in a stream on the property while drinking. The cow had seemed to be having difficulty breathing prior to this. You perform a necropsy on the cow and note perirenal edema and a hydrothorax. What do you think this cow may have eaten?

Slaked lime supplemented as 10% of a concentrate feed would have been a good preventitive measure for the cow that you found perirenal edema in on post mortem.

A horse falls ill after eating shaving from its bedding. What tree may be to blame?

The leaves and bark of black walnut trees are toxic to horses on pasture.

Treatment of clinical signs with an alpha blocker (prazosin), and/or a calcium channel blocker (nifedipine) and bute, flunixin or torbutrol would be appropriate for toxicosis from which tree?

What parts of the red maple tree can cause toxicity if ingested?

What toxic tree might you associate these clin path abnormalities with? Low PCV, hemoglobinuria, heinz bodies anemia, 10-40% methemoglobin, increased AST and SDH and azotemia

What would you use to treat the methemoglobin?